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Research 91–120 of 1000+
- The Vitamin D Receptor Is Required for Activation of cWnt and Hedgehog Signaling in Keratinocytes
- Ligand-Dependent Actions of the Vitamin D Receptor Are Required for Activation of TGF-β Signaling during the Inflammatory Response to Cutaneous Injury
- The Vitamin D Receptor as Tumor Suppressor in Skin
- Vitamin D Concentrations are Decreased in Patients with Alopecia Areata
- Role of the vitamin D receptor in hair follicle biology
- Vitamin D Receptor, UVR, and Skin Cancer: A Potential Protective Mechanism
- The hair cycle and Vitamin D receptor
- Hereditary 1,25-dihydroxyvitamin D-resistant rickets with alopecia resulting from a novel missense mutation in the DNA-binding domain of the vitamin D receptor
- Vitamin D-dependent rickets type II: extreme end organ resistance to 1,25-dihydroxy vitamin D3 in a patient without alopecia
- Reciprocal role of vitamin D receptor on β-catenin regulated keratinocyte proliferation and differentiation
- Increased prevalence of vitamin D deficiency in patients with alopecia areata: a systematic review and meta‐analysis
- Vitamin D and skin diseases: A review
- Modulatory role of vitamins A, B3, C, D, and E on skin health, immunity, microbiome, and diseases
- Tryptophan Missense Mutation in the Ligand‐Binding Domain of the Vitamin D Receptor Causes Severe Resistance to 1,25‐Dihydroxyvitamin D
- Hereditary 1,25‐Dihydroxyvitamin D–Resistant Rickets Due to an Opal Mutation Causing Premature Termination of the Vitamin D Receptor
- Vitamin D Receptor Is Required for Proliferation, Migration, and Differentiation of Epidermal Stem Cells and Progeny during Cutaneous Wound Repair
- Vitamin D resistance
- Interactions of the Vitamin D Receptor with the Corepressor Hairless
- Reduced level of 25-hydroxyvitamin D in chronic/relapsing Alopecia Areata
- Mechanisms Involved in the Relationship between Vitamin D and Insulin Resistance: Impact on Clinical Practice
- An investigation of vitamin D status in alopecia areata
- The vitamin D receptor functions as a transcription regulator in the absence of 1,25-dihydroxyvitamin D3
- Hereditary vitamin D-resistant rickets (HVDRR) owing to a heterozygous mutation in the vitamin D receptor
- Lymphoid Enhancer-binding Factor-1 (LEF1) Interacts with the DNA-binding Domain of the Vitamin D Receptor
- A unique insertion/duplication in the VDR gene that truncates the VDR causing hereditary 1,25-dihydroxyvitamin D-resistant rickets without alopecia
- Vitamin D receptor polymorphisms and the polycystic ovary syndrome: A systematic review
- Two New Unrelated Cases of Hereditary 1,25-Dihydroxyvitamin D-resistant Rickets with Alopecia resulting from the same Novel Nonsense Mutation in the Vitamin D Receptor Gene
- Evidence for 1,25-Dihydroxyvitamin D3-independent Transactivation by the Vitamin D Receptor
- Vitamin D deficiency in patients with alopecia areata: A systematic review and meta-analysis
- The Role of Vitamin D in Non-Scarring Alopecia