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Research 961–990 of 1000+
- Hereditary 1,25‐Dihydroxyvitamin D–Resistant Rickets Due to an Opal Mutation Causing Premature Termination of the Vitamin D Receptor
- Vitamin D Receptor Is Required for Proliferation, Migration, and Differentiation of Epidermal Stem Cells and Progeny during Cutaneous Wound Repair
- Vitamin D resistance
- Interactions of the Vitamin D Receptor with the Corepressor Hairless
- Reduced level of 25-hydroxyvitamin D in chronic/relapsing Alopecia Areata
- Mechanisms Involved in the Relationship between Vitamin D and Insulin Resistance: Impact on Clinical Practice
- An investigation of vitamin D status in alopecia areata
- The vitamin D receptor functions as a transcription regulator in the absence of 1,25-dihydroxyvitamin D3
- Hereditary vitamin D-resistant rickets (HVDRR) owing to a heterozygous mutation in the vitamin D receptor
- Lymphoid Enhancer-binding Factor-1 (LEF1) Interacts with the DNA-binding Domain of the Vitamin D Receptor
- A unique insertion/duplication in the VDR gene that truncates the VDR causing hereditary 1,25-dihydroxyvitamin D-resistant rickets without alopecia
- Vitamin D receptor polymorphisms and the polycystic ovary syndrome: A systematic review
- Two New Unrelated Cases of Hereditary 1,25-Dihydroxyvitamin D-resistant Rickets with Alopecia resulting from the same Novel Nonsense Mutation in the Vitamin D Receptor Gene
- Evidence for 1,25-Dihydroxyvitamin D3-independent Transactivation by the Vitamin D Receptor
- Vitamin D deficiency in patients with alopecia areata: A systematic review and meta-analysis
- The Role of Vitamin D in Non-Scarring Alopecia
- Stress-induced c-Jun-dependent Vitamin D Receptor (VDR) Activation Dissects the Non-classical VDR Pathway from the Classical VDR Activity
- Vitamin D‐dependent rickets Type II with alopecia: two case reports and review of the literature
- Vitamin D: Role in Skin and Hair
- Correlation of vitamin D and vitamin D receptor expression in patients with alopecia areata: a clinical paradigm
- The vitamin D receptor, the skin and stem cells
- Hereditary 1,25-Dihydroxyvitamin D-Resistant Rickets
- Evaluation of Keratinocyte Proliferation and Differentiation in Vitamin D Receptor Knockout Mice
- Serum vitamin D level is related to disease severity in pediatric alopecia areata
- Combined Deletion of the Vitamin D Receptor and Calcium-Sensing Receptor Delays Wound Re-epithelialization
- Hairless Modulates Ligand-Dependent Activation of the Vitamin D Receptor-Retinoid X Receptor Heterodimer
- Off-Label Uses of Topical Vitamin D in Dermatology: A Systematic Review
- The Vitamin D Receptor Is Required for Mouse Hair Cycle Progression but not for Maintenance of the Epidermal Stem Cell Compartment
- Mutations in the vitamin D receptor gene in four patients with hereditary 1,25-dihydroxyvitamin D-resistant rickets
- Estimated serum vitamin D status, vitamin D intake, and risk of incident alopecia areata among US women