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Research 601–630 of 1000+
- Hairless Suppresses Vitamin D Receptor Transactivation in Human Keratinocytes
- Vitamin D and the skin
- Novel mechanisms for the vitamin D receptor (VDR) in the skin and in skin cancer
- Functional Regulation of Adipose-Derived Stem Cells by PDGF-D
- Hereditary 1,25-Dihydroxyvitamin D Resistant Rickets due to a Mutation Causing Multiple Defects in Vitamin D Receptor Function
- Serum Ferritin and Vitamin D in Female Hair Loss: Do They Play a Role?
- Quantification of the Vitamin D Receptor−Coregulator Interaction
- Vitamin D and androgen receptor-targeted therapy for triple-negative breast cancer
- Targeted Expression of Human Vitamin D Receptor in the Skin Promotes the Initiation of the Postnatal Hair Follicle Cycle and Rescues the Alopecia in Vitamin D Receptor Null Mice
- Vitamin D‐dependent rickets type I and type II
- The Vitamin D Receptor Is Required for Activation of cWnt and Hedgehog Signaling in Keratinocytes
- Ligand-Dependent Actions of the Vitamin D Receptor Are Required for Activation of TGF-β Signaling during the Inflammatory Response to Cutaneous Injury
- The Vitamin D Receptor as Tumor Suppressor in Skin
- Vitamin D Concentrations are Decreased in Patients with Alopecia Areata
- Role of the vitamin D receptor in hair follicle biology
- Vitamin D Receptor, UVR, and Skin Cancer: A Potential Protective Mechanism
- The hair cycle and Vitamin D receptor
- Hereditary 1,25-dihydroxyvitamin D-resistant rickets with alopecia resulting from a novel missense mutation in the DNA-binding domain of the vitamin D receptor
- Vitamin D-dependent rickets type II: extreme end organ resistance to 1,25-dihydroxy vitamin D3 in a patient without alopecia
- Reciprocal role of vitamin D receptor on β-catenin regulated keratinocyte proliferation and differentiation
- Increased prevalence of vitamin D deficiency in patients with alopecia areata: a systematic review and meta‐analysis
- Vitamin D and skin diseases: A review
- Tryptophan Missense Mutation in the Ligand‐Binding Domain of the Vitamin D Receptor Causes Severe Resistance to 1,25‐Dihydroxyvitamin D
- Hereditary 1,25‐Dihydroxyvitamin D–Resistant Rickets Due to an Opal Mutation Causing Premature Termination of the Vitamin D Receptor
- Vitamin D Receptor Is Required for Proliferation, Migration, and Differentiation of Epidermal Stem Cells and Progeny during Cutaneous Wound Repair
- Vitamin D resistance
- Interactions of the Vitamin D Receptor with the Corepressor Hairless
- Reduced level of 25-hydroxyvitamin D in chronic/relapsing Alopecia Areata
- Mechanisms Involved in the Relationship between Vitamin D and Insulin Resistance: Impact on Clinical Practice
- An investigation of vitamin D status in alopecia areata