Search

everythinglearnresearchcommunity

for

Search:↓

Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.

Vitamin D receptor is important for regulating hair growth and wound healing in mice.

Targeting the Wnt pathway might help treat hair loss.

Low vitamin D receptor levels found in hair loss patients; topical vitamin D treatment suggested.

The vitamin D receptor helps regulate skin and hair health independently of its usual vitamin D ligand.

A genetic mutation caused severe rickets and alopecia in an Indian patient, but high-dose calcium and phosphate treatment improved their condition.

Vitamin D boosts a specific gene activity in kidney cells that could improve heart and kidney function.

Lack of Vitamin D receptor changes skin structure and increases certain immune cells in the skin.

As skin cells mature, vitamin D receptor levels decrease while retinoid X receptor α levels increase.

Mice without active or present vitamin D receptors maintain normal blood sugar control and islet gene expression when calcium levels are normal.

Vitamin D receptor helps control hair growth genes in skin cells.

Vitamin D receptor gene variations are not linked to alopecia areata.

A vitamin D receptor mutation causes rickets and affects immune responses.

The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.

Certain variations in the vitamin D receptor gene are linked to an increased risk of polycystic ovary syndrome.

Vitamin D receptor helps protect skin cells from UV damage and supports their growth.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.

A new gene mutation causes vitamin D-resistant rickets and hair loss in two siblings.

Certain gene variations in the Vitamin D receptor may increase the risk of chronic hair loss.

Low Vitamin D receptor levels in breast cancer are linked to worse outcomes and more bone metastases, and could be a marker for prognosis.

The study found that certain mutations in the vitamin D receptor can cause rickets and potentially affect hair growth.

Certain gene variations in the Vitamin D Receptor are linked to higher risk of female hair loss.

Vitamin D and its receptor are important for hair and scalp health and may help treat hair loss.

Vitamin D helps skin stem cells heal wounds by working with a key skin protein.

Certain gene variations may protect against skin issues and oxidative stress in women with PCOS.

Vitamin D receptor is crucial for skin wound healing.

The research showed that Vitamin D and its receptor are important for healthy bones and normal hair and skin in rats.

Gene therapy helped rats with a specific type of rickets grow hair without severe inflammation.

Gene therapy with a vitamin D receptor gene improved hair growth in rats with a type of rickets-related baldness.

Low Vitamin D and its receptors might be linked to chronic hair loss in women.