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The Rotterdam Study updated its design and objectives in 2012, providing insights into various diseases in the elderly, including skin cancer, bone health, liver disease, neurological and psychiatric conditions, and respiratory issues.

Hair loss and aging are caused by the breakdown of a key protein in hair stem cells.

Stem cells help heal skin wounds by moving and changing roles, working with other cells, and needing more research on their activation and behavior.

Women with PCOS are more likely to have impaired glucose tolerance and type 2 diabetes, especially if they are Asian or obese.

Different types of skin cells have unique genetic markers that affect how likely they are to spread cancer.

New research suggests that skin cell renewal may not require a special type of cell previously thought to be essential.

Stem cell self-renewal is complex and needs more research for full understanding.

Stem cells in the hair follicle are regulated by their surrounding environment, which is important for hair growth.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

The document concludes that while significant progress has been made in understanding skin biology and stem cells, more research is needed to fully understand their interactions with their environment.

Macrophages help regrow hair by activating stem cells using AKT/β-catenin and TNF.

PDGFA/AKT signaling is important for the growth and maintenance of certain skin fat cells.

Mice studies show that Protein Phosphatase 2A is crucial for cell growth, development, and disease prevention.

Blocking β-catenin in skin cells improves hair growth during wound healing.

The conclusion is that tissue structure is key for stem cell communication and maintaining healthy tissues.

Muscles and nerves that cause goosebumps also help control hair growth.

YAP and TAZ proteins are necessary for the development of two types of skin cancer.

The conclusion is that hair growth can be improved by activating hair cycles, changing the surrounding environment, healing wounds to create new hair follicles, and using stem cell technology.

Tissue-resident memory T cells can protect against infections and cancer but may also contribute to autoimmune diseases.

Certain immune system proteins are important for skin healing but can cause problems if there are too many of them.

Two-photon microscopy helps observe hair follicle stem cell behaviors in mice.

Activating β-catenin in different skin stem cells causes various types of hair growth and skin tumors.

The document concludes that mouse models are crucial for studying hair biology and that all mutant mice may have hair growth abnormalities that require detailed analysis to identify.

Glutamine metabolism affects hair stem cell maintenance and their ability to change back to stem cells.

Skin tumor regression is helped by retinoic acid signaling blocking Wnt signaling.

Mice can grow new hair follicles after skin wounds through a process not involving existing hair stem cells, but requiring more research to understand fully.

High lactate dehydrogenase activity is not necessary for the growth of squamous cell carcinoma.

The protein SLC1A3 is important for activating skin stem cells and is necessary for normal hair and skin growth in mice.

Meis1 is crucial for skin health and tumor development.

The document concludes that careful design of genetic fate mapping experiments is crucial for accurate cell lineage tracing in mice.