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Mutations in Gasdermin A3 cause skin inflammation and hair loss by disrupting mitochondria.

Overexpressing the mineralocorticoid receptor in mouse skin causes skin thinning, early skin barrier development, eye issues, and hair loss.

Noggin overexpression delays eyelid opening by affecting cell death and skin cell development.

α3β1-integrin is crucial for maintaining normal hair follicle shape and function but not needed for the development of the surrounding skin.

Lymphatic vessels are essential for hair follicle growth and skin regeneration.

The hair follicle pathway significantly affects how easily water-loving chemicals pass through the skin.

Activating Notch in adult skin causes T cells and neural crest cells to gather, leading to skin issues.

Mice without certain skin proteins had abnormal skin and hair development.

Fetal wounds heal without scarring because of different biological factors, which could help improve adult wound healing.

Placental growth factor may help treat hair loss.

The Ovol2-Zeb1 circuit is crucial for skin healing and hair growth by guiding cell movement and growth.

Researchers created early-stage hair-like structures from skin cells, showing how these cells can self-organize, but more is needed for complete hair growth.

The protein Par3 is crucial for healthy skin, affecting the skin barrier, cell differentiation, and stem cell maintenance.

Blocking TSLP reduces skin inflammation and cell overgrowth in psoriasis.

The photolyase-based device significantly changed the size and heat of potential skin cancer areas in patients.

Lanyu pigs show that partial-thickness wounds can partially regenerate important skin structures, which may help improve human skin healing.

Aging causes changes in the scalp that can affect hair growth and lead to older-looking hair in women.

Newborns with the common rash Erythema Toxicum have many active mast cells in their skin, but these cells don't produce the LL-37 peptide.

People with pemphigus vulgaris and pemphigus foliaceus often have smaller sebaceous glands on their scalp.

Mice with too much sPLA₂-IIA have hair loss and poor wound healing due to abnormal hair growth and stem cell depletion.

Deleting the PTEN gene in mice causes nerve cells to grow larger and heal better after injury, but may cause overgrowth and hair loss in older mice.

Dermal EZH2 controls skin cell development and hair growth in mice.

The "Two-Cell Assemblage" assay is a new, simple method to identify substances that may promote hair growth.

Scientists created a new cell line from Cashmere goat hair and found that cytokeratin 13 is a unique marker for certain skin cells.

Squarticles, tiny particles made from sebum-derived lipids, can effectively deliver minoxidil, a hair growth drug, directly to hair follicles and skin cells, with less skin penetration and more tolerability.

Artificial dermal template treatment can stimulate complete skin and hair follicle regrowth.

Too much β-catenin activity can mess up the development of mammary glands and make them more like hair follicles.

Hair follicle stem cells in hairpoor mice are disrupted, causing hair loss.

Ezh2 controls skin development by balancing signals for dermal and epidermal growth.

Scientists discovered a new way UVB light increases skin pigmentation through the ATP-P2X7 pathway.