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Low-dose radiation affects hair stem cell function and survival by changing their genetic material's structure.

Dying cells can help with faster healing and new hair growth by releasing a growth-promoting molecule.

The gene p53 is crucial for removing damaged cells to allow for healthy tissue renewal.

Different types of sun exposure can damage skin cells and affect healing, with chronic exposure being more harmful, and certain immune cells help in the repair process.

Notch1 signaling is crucial for improving wound healing and skin regeneration by affecting stem cell behavior.

Wnt10b helps blood stem cells grow after injury.

Mammals might fail to regenerate not because they lack the right cells, but because of how cells respond to their surroundings, and changing this environment could enhance regeneration.

Transit-amplifying cells are crucial for tissue repair and can contribute to cancer when they malfunction.

The African spiny mouse heals skin without scarring due to different protein activity compared to the common house mouse, which heals with scarring.

mTORC1 signaling needed for quick hair follicle recovery after radiation damage.

Lung and liver macrophages protect our tissues and their dysfunction can cause various diseases.

Zebrafish are useful for studying and developing treatments for human skin diseases.

Blocking a certain signal in the gp130 receptor can improve tissue healing and lessen osteoarthritis symptoms.

Different types of skin cells and immune cells play a role in healing UV-damaged skin, with chronic UV exposure causing lasting damage to certain skin cells.

Chemotherapy and radiation therapy cause skin and hair damage by altering gene expression and signaling pathways.

Fibroblasts, cells usually linked to tissue repair, also help regenerate various organs and their ability decreases with age. Turning adult fibroblasts back to a younger state could be a new treatment approach.

Certain cells outside the hair follicle's bulge area can quickly regenerate damaged hair follicles, potentially helping to reduce hair loss from cancer treatments.

Higher minoxidil dose helps hair growth in non-responders without side effects.

Early and late matrix progenitors in hair follicles create different cell layers, with early ones forming the companion layer and later ones forming the inner root sheath and hair shaft.

Sweat glands and hair follicles are determined by opposing signals, with BMPs promoting sweat glands and blocking BMPs leading to hair follicles.

Researchers improved a method to study individual cells in newborn mouse skin and found a way to assess the severity of a skin condition in humans.

The study found a link between the severity of Lichen Planopilaris seen by doctors and the details seen under a microscope, and created a new way to measure this severity.

Cells lacking the Bax protein can outcompete others, leading to better tissue repair and hair growth.

The JAK/STAT pathway is important in cell processes and disease, and JAK inhibitors are promising for treating related conditions.

Lacking cyclin D3 reduces skin cancer growth without affecting normal skin cell growth.

Gata6 is important for protecting hair growth cells from DNA damage and keeping normal hair growth.

Urokinase, a type of protein, helps skin cells multiply faster, especially in newborn mice.

A WNT10A gene mutation leads to ectodermal dysplasia by disrupting cell growth and differentiation.

Runx1 controls fat-related genes important for normal and cancer cell growth, affecting skin and hair cell behavior.

Eating high glycemic foods and drinking milk may worsen acne by increasing insulin and IGF-1 levels.