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Removing Mediator 1 causes teeth cells to turn into hair cells.

Changing the structure of the hair follicle's environment can affect hair quality but doesn't stop hair growth.

Certain groups of medications given when leaving the hospital are linked to a higher risk of negative drug reactions within 30 days, especially in older adults with respiratory issues.

The goal is to create a tool for patients to report their experiences with various hair and scalp disorders.

Mouse touch-sensitive nerve cells adjust their connections based on competition with other similar cells.

Satoyoshi syndrome is likely an autoimmune disease that mainly affects young women and improves with immune-related treatments.

A specific molecular switch, driven by MAPK/ERK signaling, helps spiny mice heal wounds by regenerating skin instead of forming scars.

ILC1-like cells can cause alopecia areata by attacking hair follicles.

The enzyme 5α-reductase is important for proper blood vessel development during the fertility-related transformation of the uterus lining.

Primary cilia affect the size and oil production of eye glands but not the oil's makeup.

People with blood group O may have a higher risk of developing post-COVID-19 syndrome.

Skin lesions in Carney complex are likely caused by a specific group of skin cells that promote pigment production due to a genetic mutation.

The gp130 receptor helps in tissue regeneration and disease progression, and manipulating it could improve healing and prevent disease.

4-aminopyridine helps skin wounds heal faster and better.

Special gut cells help stem cells move to and fix injured areas by activating a specific signaling pathway.

Gene therapy in mice increased lifespan and improved health without causing cancer.

The gene Tfap2b is essential for creating a type of stem cell in zebrafish that can become different pigment cells.

KIF18B is important for correctly positioning cell division machinery in skin cells, affecting hair follicle development.

Removing a specific gene in certain skin cells causes hair loss on the body by disrupting normal hair development.

A specific immune response helps control mite populations on the skin, maintaining healthy hair follicles.

Mothers have more hair proteins than their children, with age-related differences in protein patterns, and some proteins in hair could indicate early childhood development.

Stress can cause a type of hair loss in mice lacking the CCHCR1 gene.

Ezh2 controls skin development by balancing signals for dermal and epidermal growth.

Dermal EZH2 controls skin cell growth and differentiation in mice.

Glutamic acid helps mice grow hair.

Different fish use the same genes to regrow teeth.

The structure of SRD5A reveals how it reduces steroids, aiding drug design for related health conditions.

Neutrophil extracellular traps slow down hair follicle healing after injury.

Activating β-catenin increases melanocytes and decreases Schwann cells.

Lef1 helps adult skin cells maintain their ability to heal wounds and regenerate hair, but the study's methods and conclusions have been questioned.