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Research 811–840 of 822
- Cell Therapy for Androgenetic Alopecia: Elixir or Trick?
- Repurposing DPP4-Inhibition to Improve Hair Follicle Activation and Regeneration
- Revolutionizing Radiotoxicity Management with Mesenchymal Stem Cells and Their Derivatives: A Focus on Radiation-Induced Cystitis
- Mechanical force drives the initial mesenchymal-epithelial interaction during skin organoid development
- The influence of ESR1 polymorphisms on selected hormonal, metabolic and mineral balance markers in women with hyperandrogenism
- miR-29a-5p Inhibits Prenatal Hair Placode Formation Through Targeting EDAR by ceRNA Regulatory Network
- Adipogenic Differentiation of Canine Hair Follicle Stem Cells (cHFSCs)
- DataSheet1_Single-Cell Transcriptomics Reveals the Molecular Anatomy of Sheep Hair Follicle Heterogeneity and Wool Curvature.zip
- Current issues of skin aging and strategies for its correction
- Hair Follicle Stem Cells and Hair Regeneration
- Cells, growth factors and biomaterials used in tissue engineering for hair follicles regeneration
- Investigation of hub genes and immune infiltration in androgenetic alopecia using bioinformatics analysis
- Restoration of Hair Luster via Novel Biomarker COL7A1 by Minoxidil, Caffeine, and Biotin
- 688 Precise and efficient editing of the COL7A1 gene in RDEB derived iPSCs with CRISPR/Cas9 and prime editing
- Hair follicle aging is driven by transepidermal elimination of stem cells via COL17A1 proteolysis
- Expression of anti-aging type-XVII collagen (COL17A1/BP180) in hair follicle-associated pluripotent (HAP) stem cells during differentiation
- Hair follicle aging is driven by transepidermal elimination of stem cells via COL17A1 proteolysis
- An <i>mTurq2-Col4a1</i> mouse model allows for live visualization of mammalian basement membrane development
- 244 Defective maintenance of hair follicle stem cells through COL17A1 loss orchestrates the hair follicle aging program
- A Window into Mammalian Basement Membrane Development: Insights from the<i>mTurq2-Col4a1</i>Mouse Model
- Three Novel Homozygous Point Mutations and a New Polymorphism in the COL17A1 Gene: Relation to Biological and Clinical Phenotypes of Junctional Epidermolysis Bullosa
- The Genetics of Human Skin Disease
- 519 Phase I/IIa clinical trial for recessive dystrophic epidermolysis bullosa using genetically corrected autologous keratinocytes
- Read-Through for Nonsense Mutations in Type XVII Collagen‒Deficient Junctional Epidermolysis Bullosa
- CRISPR/Cas9-Mediated Generation of <i>COL7A1</i>-Deficient Keratinocyte Model of Recessive Dystrophic Epidermolysis Bullosa.
- 572 Defining chronic wound types in recessive dystrophic epidermolysis bullosa patients for clinical outcome assessment
- 312 CRISPR/Cas9-based targeted genome editing for correction of recessive dystrophic epidermolysis bullosa using iPS cells
- Comparative ubiquitinomics of human skin reveals insulin receptor ubiquitination as a regulator of collagen secretion
- Comparative ubiquitinomics of human skin reveals insulin receptor ubiquitination as a regulator of collagen secretion
- A homozygous nonsense mutation identified in <i>COL7A1</i> in a family with autosomal recessive dystrophic epidermolysis bullosa