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Research 271–300 of 666
- Dermatologic Side Effects of Systemic Targeted Anticancer Therapy
- Immunopathogenesis of Primary Cicatricial Alopecia
- Roles of MED1 in Quiescence of Hair Follicle Stem Cells and Maintenance of Normal Hair Cycling
- Targeting the Hippo/YAP/TAZ signalling pathway: Novel opportunities for therapeutic interventions into skin cancers
- Long-Term Bezafibrate Treatment Improves Skin and Spleen Phenotypes of the mtDNA Mutator Mouse
- TAZ enhances the activity of the AKT signaling pathway to promote adipogenesis of gADSCs
- Paracrine TGF-β Signaling Counterbalances BMP-Mediated Repression in Hair Follicle Stem Cell Activation
- Novel mechanisms for the vitamin D receptor (VDR) in the skin and in skin cancer
- Quantification of the Vitamin D Receptor−Coregulator Interaction
- Genetic Analysis of Androgen Receptors in Development and Disease
- A Missense Mutation within the Helix Termination Motif of KRT25 Causes Autosomal Dominant Woolly Hair/Hypotrichosis
- YAP/TAZ Signalling Controls Epidermal Keratinocyte Fate
- Involvement of mechanical stress in androgenetic alopecia
- Exploring the Role of GLP‐1 Agents in Managing Diabetic Foot Ulcers: A Narrative and Systematic Review
- Optimal Exercise Levels for Dermatology Patients With Psoriasis
- Epigenetic Regulation of Medulloblastoma Proliferation by Finasteride-Induced Modulation of BTG2 and CD244 Gene Methylation
- Androgens and the hair follicle. Cultured human dermal papilla cells as a model system.
- Physical and Functional Interaction between the Vitamin D Receptor and Hairless Corepressor, Two Proteins Required for Hair Cycling
- WNT Signaling in Disease
- RBPJ Mutations Identified in Two Families Affected by Adams-Oliver Syndrome
- Concise Review: Wnt Signaling Pathways in Skin Development and Epidermal Stem Cells
- Hairless Suppresses Vitamin D Receptor Transactivation in Human Keratinocytes
- Vitamin D and the skin
- Aldo-Keto Reductase (AKR) 1C3 inhibitors: a patent review
- 17β-estradiol Inhibits the Production of RANTES in Human Keratinocytes
- Hereditary 1,25-dihydroxyvitamin D-resistant rickets with alopecia resulting from a novel missense mutation in the DNA-binding domain of the vitamin D receptor
- Evidence for 1,25-Dihydroxyvitamin D3-independent Transactivation by the Vitamin D Receptor
- Lack of androgen receptor transcriptional activity in human keratinocytes
- THY1-mediated mechanisms converge to drive YAP activation in skin homeostasis and repair
- Absence of vitamin D receptor (VDR)‐mediated PPARγ suppression causes alopecia in VDR‐null mice