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Research 841–870 of 1000+
- Probing the Effects of Stress Mediators on the Human Hair Follicle
- Immunology of the Human Nail Apparatus: The Nail Matrix Is a Site of Relative Immune Privilege
- Platelet-Rich Plasma for Androgenetic Alopecia
- Controlled Delivery of T-box21 Small Interfering RNA Ameliorates Autoimmune Alopecia (Alopecia Areata) in a C3H/HeJ Mouse Model
- Alopecia Areata
- Alopecia areata update
- Calcitonin gene‐related peptide (CGRP) may award relative protection from interferon‐γ‐induced collapse of human hair follicle immune privilege
- Does collapse of immune privilege in the hair-follicle bulge play a role in the pathogenesis of primary cicatricial alopecia?
- Necrosis of the donor site after hair restoration with follicular unit extraction (FUE): A case report
- Health-related quality of life questionnaire for polycystic ovary syndrome (PCOSQ-50): development and psychometric properties
- Alopecia Areata
- Can the Hair Follicle Become a Model for Studying Selected Aspects of Human Ocular Immune Privilege?
- The aetiology and pathogenesis of alopecia areata
- Self Prion Protein Peptides are Immunogenic in Lewis Rats
- Exacerbation of alopecia areata during pegylated interferon alpha-2b and ribavirin therapy, possibly due to the collapse of hair follicle immune privilege
- Two mouse mutations mapped to chromosome 11 with differing morphologies but similar progressive inflammatory alopecia
- UVB-induced depletion of donor-derived dendritic cells prevents allograft rejection of immune-privileged hair follicles in humanized mice
- Alopecia areata multiplex following autologous dermal micrograft injection for treatment of androgenetic alopecia
- Case of alopecia induced by sorafenib, possible mechanism similar to alopecia areata
- The Function and Structure of the Skin
- Immune privilege and alopecia areata
- A Clinical Study of Androgenetic Alopecia (V)
- 516 Possible role of ILC1 in the pathogenesis of alopecia areata (AA)
- A Clinical Study of Androgenic Alopecia (VII)
- Resident T cell activation leads to human hair follicle immune privilege loss ex vivo, which is prevented by the DHODH inhibitor farudodstat: relevance for alopecia areata
- Image 1_Resident T cell activation leads to human hair follicle immune privilege loss ex vivo, which is prevented by the DHODH inhibitor farudodstat: relevance for alopecia areata.pdf
- Alopecia Areata: A Comprehensive Review of Pathogenesis, Diagnosis, and Emerging Therapeutics
- LB1029 The common fragrance, linalool, promotes core hair follicle pathology events associated with frontal fibrosing alopecia through activating a cognate olfactory receptor in the bulge
- 1419 Pre-clinical in-vitro assays for high throughput screening of drug candidates for protection and restoration of immune privilege in alopecia areata
- 1432 IL-9/IL-9R signaling as a novel regulator of human hair follicle growth and immune privilege