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Epigenetic changes are crucial for stem cell behavior in skin wound healing and their disruption may lead to cancer.

The research found that the gene activity in mouse skin stem cells changes significantly as they age.

DNA methylation is essential for skin and hair follicle development, and could be a target for treating skin diseases.

Stem cells help heal skin wounds by moving and changing roles, working with other cells, and needing more research on their activation and behavior.

Different types of skin cells play specific roles in development, healing, and cancer.

Aging causes skin stem cells to work less effectively.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

The telogen phase of hair growth is active and important for preparing hair follicles for regeneration, not just a resting stage.

We need more research to better understand human hair follicle stem cells for improved treatments for hair loss and skin cancer.

Muscles and nerves that cause goosebumps also help control hair growth.

YAP and TAZ proteins are necessary for the development of two types of skin cancer.

Exosomes from dermal papilla cells help hair growth by making hair follicle stem cells multiply and change.

Melanocyte development from neural crest cells is complex and influenced by many factors, and better understanding could help treat skin disorders.

The study showed that hair follicle stem cells can maintain and organize themselves in a lab setting, keeping their ability to renew and form hair and skin.

Researchers found 15 new genetic links to skin traits in Japanese women.

Older skin has higher cancer risk due to inflammation and stem cell issues.

The vitamin D receptor is essential for skin stem cells to grow, move, and become different cell types needed for skin healing.

Transit-amplifying cells are crucial for tissue repair and can contribute to cancer when they malfunction.

Smad1 and Smad5 are essential for hair follicle development and stem cell sleepiness.

The conclusion is that skin and hair patterns are formed by a mix of cell activities, molecular signals, and environmental factors.

Vav2 and Vav3 proteins help control skin stem cell numbers and activity in both healthy and cancerous cells.

Different types of skin stem cells can change and adapt, which is important for developing new treatments.

GRK2 is essential for healthy hair follicle function, and its absence can lead to hair loss and cysts.

The research found that people's hair proteins vary, especially by ethnicity and body part, which could help identify individuals in forensic science.

The research found that people's hair proteins vary by individual and body part, with some differences between ethnic groups, which could help in forensics.

Overexpression of sPLA2-IIA in mouse skin reduces hair stem cells and increases cell differentiation through JNK/c-Jun pathway activation.

Enzymes called PADIs play a key role in hair growth and loss.

lncRNA2919 slows down rabbit hair growth by stopping cell growth and causing cell death.

DNA methylation controls lncRNA2919, which negatively affects hair growth.

Lower SMAD2/3 activation predicts more severe skin cancer.