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Human amniotic membrane helps heal skin wounds faster and with less scarring.

Transit-amplifying cells are crucial for tissue repair and can contribute to cancer when they malfunction.

Cellular senescence has both cancer-blocking and cancer-promoting effects, and targeting senescent cells may improve health and lifespan.

Prominin-1 expressing cells in the dermal papilla help regulate hair follicle size and communication but don't aid in skin repair.

Lanyu pigs show that partial-thickness wounds can partially regenerate important skin structures, which may help improve human skin healing.

Polycomb Repressive Complex 2 is not needed for hair regeneration.

Dermal papilla cells are key for hair growth and color, influencing hair type and size, and their interaction with stem cells could help treat hair loss and color disorders.

Jagged-1 in skin Tregs is crucial for timely wound healing by recruiting specific immune cells.

Scientists created a tiny, 3D model of a hair follicle that grows and acts like a real one.

Progress has been made in skin and nerve regeneration, but more research is needed to improve methods and ensure safety.

Skin health and repair depend on the signals between skin stem cells and their surrounding cells.

The right cells and signals can potentially lead to scarless wound healing, with a mix of natural and external wound healing controllers possibly being the best way to achieve this.

The research found that specific stem cells maintain skin gland openings and that disrupting their activity can cause blockages or early cancer signs, indicating a need for targeted treatments.

The extracellular matrix affects where tumors can start in the body.

Skin wounds can create fat cells that help regenerate hair follicles, with BMP signaling playing a crucial role in this process.

High levels of ERK activity are key for tissue regeneration in spiny mice, and activating ERK can potentially redirect scar-forming healing towards regenerative healing in mammals.

Telomerase-positive cells are mainly in the bulb matrix and outer root sheath of hair follicles.

Fibroblasts are crucial for tissue repair and inflammation, and understanding them can help treat fibrotic diseases.

Hair growth phase and certain genes can speed up wound healing, while an inflammatory mediator can slow down new hair growth after a wound. Understanding these factors can improve tissue regeneration during wound healing.

Notch signalling helps skin cells differentiate and prevents tumors.

Dermal fibroblasts have adjustable roles in wound healing, with specific cells promoting regeneration or scar formation.

Fat cells in the skin help start healing and form important repair cells after injury.

Scarless healing is complex and influenced by genetics and environment, while better understanding could improve scar treatment.

Feather patterns form through genetic and epigenetic controls, with cells self-organizing into periodic patterns.

Skin fat has important roles in hair growth, skin repair, immune defense, and aging, and could be targeted for skin and hair treatments.

Using the drugs AMD3100 and Tacrolimus together greatly improves skin healing and hair growth after a deep skin cut by increasing stem cells in the wound.

Hair follicle regeneration possible, more research needed.

15-lipoxygenase helps keep skin healthy by reducing inflammation.

Researchers created a rat model to study skin damage caused by radiation, which could help develop new treatments.

Sebaceous glands can help harvest hair follicle stem cells to regenerate skin and hair.