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Different signals work together to change gene activity and guide hair follicle stem cells to become specific cell types.

Melanocyte development from neural crest cells is complex and influenced by many factors, and better understanding could help treat skin disorders.

Gene therapy, especially using atoh1, shows promise for creating functional sensory hair cells in the inner ear, but dosing and side effects need to be managed for clinical application.

Jasmonic acid helps plants grow, defend against threats, and survive stressful conditions like drought and salt.

Wnt/beta-catenin signaling in the skin helps fat cell development during hair growth and repair.

The Hedgehog signaling pathway is important for skin and hair growth and can lead to cancer if it doesn't work right.

The FOXN1 gene is crucial for developing immune cells and preventing immune disorders.

RACB in barley is crucial for cell polarity and nucleus positioning, aiding fungal infection.

Androgens affect bone and fat cell development differently based on the cells' embryonic origin.

Poplar seed hairs grow from the placenta at the ovary base, with endoreduplication playing a key role in their development, and share similar cellulose synthesis processes with cotton fibers.

The Notch signaling pathway is important for hair follicle development and could help create treatments for hair disorders.

Patients with certain FoxN1 gene mutations have severe immune issues but normal skin and hair.

miR-218-5p helps skin and hair growth by targeting SFRP2 and activating a specific signaling pathway.

The nervous system helps control stem cell behavior and immune responses, affecting tissue repair and maintenance.

Deregulation of stem cell function is central to the development of some cancers.

The Wnt/β-catenin pathway is important for tissue development and has potential in regenerative medicine, but requires more research for therapeutic use.

Noggin overexpression delays eyelid opening by affecting cell death and skin cell development.

FoxN1 overexpression in young mice harms immune cell and skin development.

The Hairless gene is crucial for hair cell development, affecting whether skin cells become hair or skin and oil gland cells.

Recent findings suggest that genetic factors, immune system issues, and skin cell defects might contribute to the development of hidradenitis suppurativa.

Certain miRNAs are linked to chicken feather development.

Not having enough or having too much of the protein Grainyhead-like 3 leads to various developmental problems.

Activin A increases inner ear hair cell development, while follistatin decreases it.

Fibroblast growth factor receptor signaling controls cell development and repair, and its malfunction can cause disorders and cancer, but it also offers potential for targeted therapies.

EGF and KGF signalling prevent hair follicle formation and promote skin cell development in mice.

Researchers identified specific genes that are important for mouse skin cell development and healing.

Keratin genes change gradually during skin cell development and should be used carefully as biomarkers.

HtrA2 activity is crucial for normal hair growth by regulating fat cell development.

Activating β-catenin increases melanocytes and decreases Schwann cells.

ARL15 is important for fat cell development and the release of the hormone adiponectin.