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Tiny particles from stem cells help activate hair growth cells and encourage hair growth in mice without being toxic.

Applying a special compound can promote hair growth without harmful side effects.

KGF and activin are crucial for skin healing and repair.

Too much Smad7 can cause serious changes in skin tissues, including problems with hair growth, thymus shrinkage, and eye development issues.

Hair color production is closely linked to the active growth phase of hair in mice and may also influence hair growth itself.

Stress increases a factor in mice that leads to hair loss, and blocking this factor may prevent it.

Overexpressing Dsg3 in mice skin causes excessive cell growth and abnormal skin development.

PDGFA/AKT signaling is important for the growth and maintenance of certain skin fat cells.

New single-cell analysis techniques are improving our understanding of stem cells and could help in treating diseases.

Skin-derived precursors in hair follicles come from different origins but function similarly.

Activating TRPV3 stops human hair growth.

Formyl-peptide receptor agonists could be new anti-inflammatory drugs.

The TGM3 gene's promoter region is key for skin and hair cell function and may aid gene therapy.

Olive oil compounds may help prevent cancer in animals, but human results are mixed.

Lampreys have a functional vitamin D receptor that may help detoxify harmful substances.

FGF signaling is a promising target for developing treatments for wounds, metabolic diseases, and cancer.

MicroRNA-214 is important for skin and hair growth because it affects the Wnt pathway.

Both genes and environmental factors like chemicals may contribute to the increase in hypospadias, but the exact causes are still unclear.

One minoxidil-sensitive potassium channel exists in human hair follicles.

A mother's PPARγ is crucial for preventing harmful milk that can cause inflammation and growth problems in babies.

Scientists turned mouse stem cells into skin cells that can grow into skin layers and structures.

5α-reductase is essential for male sexual development and its inhibitors have potential in treating various conditions related to hormone action.

Cystatin M/E strongly inhibits cathepsin V and cathepsin L, important for skin formation.

The study found that two enzymes linked to hair loss are located in different parts of the scalp, supporting a common treatment's effectiveness.

Finasteride effectively blocks rat enzymes, but with varying methods and strength.

A genetic change in the EDAR gene causes the unique hair traits found in East Asians.

Mutant CDP/Cux protein causes hair defects and reduced male fertility in mice.

A WNT10A gene mutation leads to ectodermal dysplasia by disrupting cell growth and differentiation.

The p75 neurotrophin receptor is important for hair follicle regression by controlling cell death.

Mutations in the PADI3 gene are linked to a higher risk of scarring hair loss in women of African descent.