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A chemical called 5-Bromo-2′-deoxyuridine caused rapid hair loss in mice by killing certain skin cells through a specific cell death pathway.

Epidermal keratinocytes start wound healing and inflammation after schistosome infection.

Deleting the p63 gene in certain cells causes problems in thymus development and severe hair loss in mice.

Keratin 18 helps diagnose and predict cancer progression and affects cancer growth and spread.

A protein called EGFR protects hair follicle stem cells, and when it's disrupted, hair follicles can be damaged, but blocking certain pathways can restore hair growth.

A specific immune response helps control mite populations on the skin, maintaining healthy hair follicles.

Non-immune dermal cells dominate, epidermal cells increase after day 9, and certain immune cells persist beyond inflammation in wound-induced hair follicle regeneration.

Keeping β-catenin levels high in mammary cells disrupts their development and branching.

The enzyme sEH is important for hair growth and its inhibition could help treat hair loss.

Thymic mesenchymal cells have unique gene expression that supports their specific functions in the thymus.

Blocking DPP4 can potentially speed up hair growth and regeneration, especially after injury or in cases of hair loss.

Human skin xenografting could improve our understanding of skin development, renewal, and healing.

The research identified two types of keratinocytes in chicken scales: one for hard scales and another for soft skin, with similarities to human skin differentiation.

The document concludes that immune system abnormalities cause alopecia areata, but the exact process is still not completely understood.

Fibroblast state switching is crucial for skin healing and development.

Foxn1 is important for fat development, metabolism, and wound healing in skin.

Scarless healing is complex and influenced by genetics and environment, while better understanding could improve scar treatment.

The technique effectively shows how human skin and hair cells form into ball-like structures.

Keratinocytes help heal skin wounds by interacting with immune cells and producing substances that kill pathogens.

Keratin 79 marks a new group of cells that are key for creating and repairing the hair follicle's structure.

The document concludes that better targeted treatments are needed for wound healing, and single-cell technologies may improve cell-based therapies.

The study found that stem cells and neutrophils are important for regenerating hair follicle structures in mice.

Free fatty acids in sebum boost skin's defense against acne by increasing antimicrobial peptides.

Autophagy helps control skin inflammation and cancer responses and regulates hair growth by affecting stem cell activity.

Mesenchymal stem cell proteins in a special gel improved healing of severe burns.

Hair follicles help attract immune cells to the skin during stress.

Activating β-catenin in different skin stem cells causes various types of hair growth and skin tumors.

Androgen receptor activity blocks Wnt/β-catenin signaling, affecting hair growth and skin cell balance.

The congress highlighted new gene therapy techniques and cell transplantation methods for treating diseases.

The skin has a complex immune system that is essential for protection and healing, requiring more research for better wound treatment.