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NIR-II imaging effectively tracked stem cells that helped repair facial nerve defects in rats.

Prdm1 is necessary for early whisker development in mice but not for other hair, and its absence changes nerve and brain patterns related to whiskers.

MicroRNA-302 helps improve the conversion of body cells into stem cells by blocking NR2F2.

A 55-year-old woman with several health conditions did not see hair regrowth after a transplant.

The circadian clock affects skin stem cell behavior, impacting aging and cancer risk.

Hair transplant surgery may cause alopecia areata, a new possible complication.

Fat-related cells are important for initiating hair growth.

Different types of skin cells have unique genetic markers that affect how likely they are to spread cancer.

Human dermal fibroblasts are the main cells targeted by a virus that can cause a deadly skin cancer, and a certain inhibitor can effectively block this infection.

Activating Nrf2 protects human hair follicles from oxidative stress and helps prevent hair growth inhibition.

The document concludes that understanding hair follicle cell cycles is crucial for hair growth and alopecia research, and recommends specific techniques and future research directions.

VEGF stimulates hair cell growth and increases growth receptor levels through a specific signaling pathway.

Mice genetically modified to produce more CD109 in their skin had less inflammation and better healing with less scarring.

Fat-derived stem cells can help protect and repair skin stem cells from aging caused by UV light.

Notch1 signaling is crucial for improving wound healing and skin regeneration by affecting stem cell behavior.

CD201+ fascia progenitors are essential for wound healing and could be targeted for treating skin conditions.

Vav2 and Vav3 proteins help control skin stem cell numbers and activity in both healthy and cancerous cells.

Hair transplants for male baldness can show inflammation and fibrosis, but more research is needed to confirm LPP presence.

The study identified unique genes in hair follicle cells and their environment, suggesting these genes help organize cells for hair growth.

Overactivating Hedgehog signaling makes hair follicle cells in mice grow hair faster and create more follicles.

The gel with icariin speeds up wound healing, reduces scarring, and helps hair growth by controlling BMP4 signaling. It also reduces inflammation and improves wound quality in mice, adapts to different wound shapes, and gradually releases icariin to aid healing. It also prevents too much collagen and myofibroblast formation during skin healing.

Autophagy prevents early aging and maintains lipid and pheromone balance in mouse glands.

Colostrum-derived exosomes can promote hair growth and may be a promising treatment for hair loss.

Adding insulin-like growth factor 1 and bone marrow-derived stem cells to a collagen-chitosan scaffold helps wounds heal faster and regrows hair follicles.

A protein called desmoglein 3 is important for keeping hair follicle stem cells inactive and helps in their regeneration.

Wnt4 protein makes the outer skin layer thicker in burn wounds by turning on a specific healing pathway and loosening the connections between skin cells.

Foxn1 is important for fat development, metabolism, and wound healing in skin.

GPR40 agonists help hair growth through the protein ANGPTL4.

Fetal scalp cells have more regenerative genes than adult cells, and decellularized muscle matrix is better for muscle repair than commercial alternatives.

PTHrP and its receptor can control blood vessel growth and hair development in mouse skin.