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Research 121–150 of 1000+
- Stress Inhibits Hair Growth in Mice by Induction of Premature Catagen Development and Deleterious Perifollicular Inflammatory Events via Neuropeptide Substance P-Dependent Pathways
- Natural Scalp Hair Regression in Preclinical Stages of Male Androgenetic Alopecia and Its Reversal by Finasteride
- Neuroimmunology of Stress: Skin Takes Center Stage
- Keratin 17 modulates hair follicle cycling in a TN<i>Fα</i>-dependent fashion
- Abstract 1219: Defective catagen entry in EGFR deficient skin precedes cutaneous inflammation
- A role for p75 neurotrophin receptor in the control of apoptosis‐driven hair follicle regression
- p53 Involvement in the Control of Murine Hair Follicle Regression
- Expression of Cyclooxygenase Isozymes During Morphogenesis and Cycling of Pelage Hair Follicles in Mouse Skin: Precocious Onset of the First Catagen Phase and Alopecia upon Cyclooxygenase-2 Overexpression
- Wnt10b Promotes Differentiation of Mouse Hair Follicle Melanocytes
- Follicular streamers (stelae) in scarring and non-scarring alopecia
- Estrogen Leads to Reversible Hair Cycle Retardation through Inducing Premature Catagen and Maintaining Telogen
- Hair Growth Cycle Affects Hair Follicle Destruction by Ruby Laser Pulses
- Regulation of hair follicle development by exosomes derived from dermal papilla cells
- Resting no more: re‐defining telogen, the maintenance stage of the hair growth cycle
- Principles of Hair Cycle Control
- Adenovirus-Mediated Wnt10b Overexpression Induces Hair Follicle Regeneration
- Hair physiology and its disorders
- Catagen in the hairless house mouse
- Polygonum multiflorum extract support hair growth by elongating anagen phase and abrogating the effect of androgen in cultured human dermal papilla cells
- Ginsenoside Re prevents 3-methyladenine-induced catagen phase acceleration by regulating Wnt/β-catenin signaling in human dermal papilla cells
- A patient with diffuse hair loss
- Do Hair Bulb Melanocytes Undergo Apotosis During Hair Follicle Regression (Catagen)?
- Human Scalp Hair Follicles Are Both a Target and a Source of Prolactin, which Serves as an Autocrine and/or Paracrine Promoter of Apoptosis-Driven Hair Follicle Regression
- Neurogenic Inflammation in Stress-Induced Termination of Murine Hair Growth Is Promoted by Nerve Growth Factor
- A Guide to Assessing Damage Response Pathways of the Hair Follicle: Lessons From Cyclophosphamide-Induced Alopecia in Mice
- Mast cell deficient and neurokinin-1 receptor knockout mice are protected from stress-induced hair growth inhibition
- Type II Epithelial Keratin 6hf (K6hf) Is Expressed in the Companion Layer, Matrix, and Medulla in Anagen-Stage Hair Follicles
- Involvement of the Edar Signaling in the Control of Hair Follicle Involution (Catagen)
- <i>Igf1r</i> signalling acts on the anagen‐to‐catagen transition in the hair cycle
- Protective effect of Korean Red Ginseng against chemotherapeutic drug-induced premature catagen development assessed with human hair follicle organ culture model