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Fibroblast Growth Factor-9 (FGF-9) can help improve heart function in diabetic mice after a heart attack by reducing inflammation and harmful changes to the heart's structure.

Testosterone reduces heart damage and inflammation after injury.

Finasteride improves heart function and repairs damage after heart attack in mice.

A specific RNA, circNlgn, contributes to heart damage and scarring caused by the cancer drug doxorubicin.

TRPV4 helps cells repair tissue and reduce scarring by controlling calcium levels.

Finasteride helps reduce heart issues and improves heart function.

New drugs for heart disease may be developed from molecules secreted by stem cells.

When treating older patients with both heart valve disease and heart muscle disease, doctors should create personalized treatment plans that address both conditions.

Women generally handle heart enlargement better than men, but it's riskier for them if it occurs; hormones like estrogen offer some protection.

Researchers created a model to understand heart aging, highlighting the role of microRNAs and identifying key genes and pathways involved.

Higher DHT levels are linked to worse heart changes in severe aortic valve stenosis patients.

Healing of heart and skin wounds in animals are similar.

Finasteride worsens heart damage from nandrolone decanoate but protects against cell damage.

A new technology showed that the SOX9 gene might control heart scar formation after injury, suggesting new treatment possibilities.

NF-κB is crucial for zebrafish heart repair, affecting heart cell growth and repair processes.

Finasteride helps reduce heart enlargement in heart failure patients.

Male hormones may play a role in the development of heart disease, and more research is needed to understand their effects.

The secretome from mesenchymal stem cells is a promising treatment that may repair tissue and avoid side effects of stem cell transplantation.

Regulatory T-cells are important for healing and regenerating tissues in various organs by controlling immune responses and aiding stem cells.

The Wnt/β-catenin pathway is important for tissue development and has potential in regenerative medicine, but requires more research for therapeutic use.

Testosterone helps prevent skin damage in males by acting through both estrogen and androgen pathways.

Fibroblast Growth Factor-9 helps repair heart damage after a heart attack by creating new blood vessels, especially in diabetics.

Long-haul COVID can cause lasting symptoms affecting many body systems and may be linked to ongoing inflammation and immune system issues.

TLR4 is important in aging-related diseases and could be a new treatment target.

Wnt signaling may be linked to heart diseases in aging and could be a target for future treatments.

ECM-inspired wound dressings can help heal chronic wounds by controlling macrophage activity.

Finasteride may improve stem cell therapy for heart attacks by increasing cell survival.

The secretions of mesenchymal stem cells could be used for healing without using the cells themselves.

Men are more likely to have severe COVID-19 cases and fatalities than women due to factors like lifestyle, aging, and biological differences.

The Wnt/β-catenin pathway helps tissue regeneration but can also cause fibrosis, and drugs that inhibit this pathway may aid in healing skin and heart tissues.