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Research 61–90 of 1000+
- 6874 A Rare Case Of Hereditary 1,25 (OH)2D Resistant Rickets
- PO-0071 Role Of Growth Hormone Therapy In Clinical, Laboratory, Radiological Improvement Of Children With Vitamin-d Dependent Rickets Type-2
- Alopecia with Vitamin D-Dependent Rickets Type 2 A: A Case Report
- 780 Novel mechanism of UVB-mediated pigmentation through the axis of ATP-P2X7
- 丹蒽醌對人類惡性黑色素瘤細胞株(A375.S2)誘發細胞週期停滯及細胞凋亡之影響
- The impact of VDR expression and regulation in vivo
- Pathogenesis and management of TRPV3-related Olmsted syndrome
- 870 Y27632 promotes proliferation via EGFR signaling in a newly isolated and characterized human primary sebocyte cell line
- 1322 Molecular profiling of frontal fibrosing alopecia (FFA) reveals TH1 and JAK-STAT up-regulation with no suppression of hair keratins
- Additional file 10 of Time-course RNA-seq analysis reveals stage-specific and melatonin-triggered gene expression patterns during the hair follicle growth cycle in Capra hircus
- A rare case of Vitamin D dependent rickets type II: a case report
- The hair cycle and Vitamin D receptor
- Vitamin D Receptor: Key Roles in Bone Mineral Pathophysiology, Molecular Mechanism of Action, and Novel Nutritional Ligands
- A Study of the Expression of Small Conductance Calcium-Activated Potassium Channels (SK1-3) in Sensory Endings of Muscle Spindles and Lanceolate Endings of Hair Follicles in the Rat
- Plant elicitor Peptides regulate root hair development in Arabidopsis
- Vitamin D resistance
- Hereditary 1,25-Dihydroxyvitamin D-Resistant Rickets
- Vitamin D receptor cross-talk with p63 signaling promotes epidermal cell fate
- Structural Analyses of the Transient Receptor Potential Channels TRPV3 and TRPV6
- Ligand-Independent Actions of Vitamin D Receptor
- Normalization of Mineral Ion Homeostasis by Dietary Means Prevents Hyperparathyroidism, Rickets, and Osteomalacia, But Not Alopecia in Vitamin D Receptor-Ablated Mice<sup>1</sup>
- Normalization of Mineral Ion Homeostasis by Dietary Means Prevents Hyperparathyroidism, Rickets, and Osteomalacia, But Not Alopecia in Vitamin D Receptor-Ablated Mice
- SUN-332 A Rare Case Of Hereditary 1,25 (OH)2D Resistant Rickets
- Detection of Functionally Active Melanocortin Receptors and Evidence for an Immunoregulatory Activity of α-Melanocyte-Stimulating Hormone in Human Dermal Papilla Cells
- Mutations in the vitamin D receptor gene in four patients with hereditary 1,25-dihydroxyvitamin D-resistant rickets
- Mechanism of Vitamin D Receptor Action
- Hereditary 1,25-dihydroxyvitamin D-resistant rickets with alopecia resulting from a novel missense mutation in the DNA-binding domain of the vitamin D receptor
- Vitamin D Receptor Is Required for Proliferation, Migration, and Differentiation of Epidermal Stem Cells and Progeny during Cutaneous Wound Repair
- Clinical and Genetic Characterization of Tunisian Children with Hereditary 1,25-Dihydroxyvitamin D-Resistant Rickets
- Mediator1: An Important Intermediary of Vitamin D Receptor–Regulated Epidermal Function and Hair Follicle Biology