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Research 30 of 193
- Simulative Evaluation of Taurine Against Alopecia Caused by Stress in Caenorhabditis elegans
- Ginseng volatile oil prolongs the lifespan and healthspan of Caenorhabditis elegans
- Live-cell Imaging and Analysis of Germline Stem Cell Mitosis in Caenorhabditis elegans
- <i>De Novo</i> Design of Anti-Aging Peptides
- Modulating Aging and Aging-related Symptoms Using Endogenous Small Molecules
- Efficacy of derinat as a treatment for murine and androgenetic alopecia (AGA) patients
- Phytochemicals of Alpinia zerumbet: A Review
- Do Changes in Chromosomes Cause Aging?
- Live imaging of stem cells: answering old questions and raising new ones
- Comparative biology of tissue repair, regeneration and aging
- Regulation of stem cell function by protein ubiquitylation
- Defining dermal adipose tissue
- Concise Review: The Plasticity of Stem Cell Niches: A General Property Behind Tissue Homeostasis and Repair
- Stem Cells in the Wild: Understanding the World of Stem Cells through Intravital Imaging
- Dermal papilla regulation of hair growth and pigmentation
- Wnt signaling: the good and the bad
- Stem Cells and the Niche: A Dynamic Duo
- Aging and Immortality: Quasi-Programmed Senescence and Its Pharmacologic Inhibition
- Apoptosis in human skin development: Morphogenesis, periderm, and stem cells
- Transforming growth factor-β in stem cells and tissue homeostasis
- A matter of life and death: self‐renewal in stem cells
- Exosomes for repair, regeneration and rejuvenation
- Lignans and Neolignans: Plant secondary metabolites as a reservoir of biologically active substances
- Targeting Wnt/β-Catenin Pathway for Developing Therapies for Hair Loss
- TGF-β Family Signaling in Epithelial Differentiation and Epithelial–Mesenchymal Transition
- Hardwiring Stem Cell Communication through Tissue Structure
- PAK4 signaling in health and disease: defining the PAK4–CREB axis
- Ferreting out stem cells from their niches
- Beyond Expectations
- Transient mitochondrial DNA double strand breaks in mice cause accelerated aging phenotypes in a ROS-dependent but p53/p21-independent manner