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Human placental extract and minoxidil together significantly promote hair growth.

The Wnt/β-catenin pathway helps tissue regeneration but can also cause fibrosis, and drugs that inhibit this pathway may aid in healing skin and heart tissues.

Hair loss treatments work better with lifestyle changes.

Human placental extract may help hair growth by affecting certain cell signals and could be more effective with minoxidil.

Claudin-1 and Claudin-3 are crucial for keeping hair follicle structure and preventing a type of hair loss called telogen effluvium.

Prostasin has two roles in skin: one for normal skin development without needing activation, and another for proper hair growth that requires activation.

Lichen planopilaris may be an autoimmune disease causing hair loss due to immune system issues in hair follicles.

GDNF helps grow hair and heal skin wounds by acting on hair stem cells.

Lymphatic vessels are essential for hair follicle growth and skin regeneration.

Minoxidil nanoparticles significantly boost hair growth in mice compared to regular minoxidil.

Both human platelet lysate and minoxidil can promote hair growth, but they affect different genes and cell survival rates.

Activating Nrf2 helps wounds heal faster by increasing hair follicle stem cells.

Certain signals and genes play a key role in hair growth and regeneration, and understanding these could lead to new treatments for skin regeneration.

Activating Nrf2 protects human hair follicles from oxidative stress and helps prevent hair growth inhibition.

The study suggests that the arrector pili muscle is important for hair health and its damage might contribute to hair loss.

Patient-derived melanocytes can potentially treat vitiligo by restoring skin pigmentation.

The we/we wal/wal mice have defects in hair growth and skin layer formation, causing hair loss, useful for understanding alopecia.

Disrupting Stat3 in hair follicle stem cells greatly reduces skin tumor formation.

Hair follicles offer promising targets for delivering drugs to treat hair and skin conditions.

Both Th1 and Th2 immune responses are increased in alopecia areata, with Th2 response more strongly linked to how severe the disease is.

Steroid sex hormones activate matriptase in prostate cancer cells but not in breast cancer cells.

New hair can grow from large wounds in mice, but less so as they age, involving reprogramming of skin cells and specific molecular pathways.

PGD2 increases androgen receptor activity in hair cells, which could be targeted to treat hair loss.

Sunlight worsens hair loss; protect scalp.

VEGFR-2 activation is likely involved in hair follicle growth, survival, and development.

Metformin, usually used for diabetes, can also help treat hair loss from alopecia areata due to its ability to reduce inflammation and stimulate new hair growth.

Light-based treatment, Photobiomodulation, shows promise for non-invasive skin therapy with few side effects.

Certain inhibitors applied to the skin can promote hair growth by maintaining a key hair growth signal.

Lactate production is important for activating hair growth stem cells.

Hair follicle bulge cells don't help skin regrow after glucocorticoid damage; interfollicular epidermis cells do.