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Disruptions in hair follicle fibroblast dynamics can cause hair growth problems.

FA2H is essential for normal fur and sebum production in mice.

Vitamin D receptor is crucial for starting hair growth after birth.

Indian Hedgehog helps control skin cell growth and protects against aggressive skin cancer.

Cynomorium songaricum has many health benefits and could help with hair regrowth.

Disrupted stem cell signals in hairpoor mice cause hair loss.

Foxn1 is important for fat development, metabolism, and wound healing in skin.

Vitamin D receptor is important for regulating hair growth and wound healing in mice.

Trapa bispinosa Roxb. extract may help reduce prostate size and cell growth in BPH.

BMAL1 controls skin cell growth and UV damage risk, peaking at night.

Lacking cyclin D3 reduces skin cancer growth without affecting normal skin cell growth.

Overexpressing follistatin in mice delays wound healing and reduces scar size.

Thyroid hormone receptors are essential for hair growth and wound healing.

A mouse gene mutation increases the risk of skin cancer.

Hormones and stretching both needed for nipple area skin growth in mice.

The type 3 IP3 receptor is important for controlling hair loss and growth.

Mirex seems to promote a unique group of skin cells different from those affected by another tumor promoter, TPA.

Notch1 signaling is crucial for improving wound healing and skin regeneration by affecting stem cell behavior.

Allopregnanolone increases growth and changes gene activity in human brain cancer cells.

β-Catenin signaling is involved in brain cell growth after injury and could be a therapy target.

The engineered skin substitute helped grow skin with hair on mice.

HPV genes and estradiol increase a cancer-related signaling pathway, which may be targeted for cervical cancer treatment.

A small group of slow-growing cells causes basal cell carcinoma to return after treatment.

Wnt and Notch signaling help wound healing by promoting cell growth and regulating cell differentiation.

Endoglin is crucial for proper hair growth cycles and stem cell activation in mice.

A chemical called 5-Bromo-2′-deoxyuridine caused rapid hair loss in mice by killing certain skin cells through a specific cell death pathway.

Mouse hair follicle stem cells lose their ability to change into different cell types after being grown for a long time.

Wnt proteins from certain skin cells are crucial for normal hair growth and renewal.

Disrupting Smad4 in mouse skin causes early hair follicle stem cell activity that leads to their eventual depletion.

Stem cells regenerate tissues and their behavior varies by environment, suggesting the hematopoietic system model may need revision.