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miR-22, a type of microRNA, controls hair growth and its overproduction can cause hair loss, while its absence can speed up hair growth.

Frizzled 3 and Frizzled 6 together control the orientation of mouse hair follicles.

A new painless method to collect hair follicles helps study DNA damage and aging.

New cell-based therapies may improve hair loss treatments in the future.

Balding might help identify men at higher risk for severe COVID-19, but more research is needed.

Aged individuals heal wounds less effectively due to specific immune cell issues.

The 3D-SeboSkin model effectively simulates Hidradenitis suppurativa and is useful for future research.

Cathepsin L is essential for normal hair growth and development.

Hair color production is closely linked to the active growth phase of hair in mice and may also influence hair growth itself.

LEF1 interacts with Vitamin D Receptor, affecting hair follicle regeneration and this could be linked to hair loss conditions.

GMG-43AC may help reduce unwanted hair growth and treat certain hair loss conditions.

Blocking Wnt/β-catenin signaling with EGF receptor is necessary for proper hair growth.

Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.

Hair in mammals likely evolved from glandular structures, not scales.

Gamma-rays exposure during the resting phase of hair growth can damage hair regeneration and color in mice.

Cold atmospheric plasma may speed up wound healing and control infections.

BMP signaling is essential for the development of touch domes.

A specific hair protein variant increases the spread of breast cancer and is linked to worse survival rates.

Blocking testosterone speeds up wound healing in males.

Intu gene is crucial for hair follicle formation by helping keratinocytes differentiate through primary cilia.

Epithelial-mesenchymal interactions control hair growth cycles through specific molecular signals.

Autophagy is crucial for normal sebaceous gland function and sebum composition.

The study concluded that a protein important for hair strength is regulated by certain molecular processes and is affected by growth phases.

Enterococcus faecalis delays wound healing by disrupting cell functions and creating an anti-inflammatory environment.

Estrogens and progesterone are key in regulating melasma pigmentation.

Hair follicle melanocytes die during hair regression.

Dandruff might be caused by changes in how hair follicles naturally release oils and an immune response to this imbalance.

Hair keratins evolved from ancient proteins, diversifying through gene changes, crucial for forming claws and later hair in mammals.

Certain natural and synthetic compounds may help treat inflammatory skin diseases by targeting a specific signaling pathway.

Delta-opioid receptors affect skin cell circadian rhythms, possibly impacting wound healing and cancer.