Search

everythinglearnresearchcommunity

for

Search:↓

Estrogens significantly influence hair growth by interacting with receptors in hair follicles and may help regulate the hair growth cycle.

Fetal wound healing changes with development, affecting inflammation and collagen, which may influence scarring.

All-trans retinoic acid causes hair loss by increasing TGF-β2 in hair follicle cells.

Dihydrotestosterone increases certain inflammatory signals in skin cells, potentially contributing to acne.

Retinoids increase steroid sulfatase activity in leukemia cells through RARα/RXR and involves certain pathways like phosphoinositide 3-kinase and ERK-MAP kinase.

Researchers found a genetic region that influences the number of coat layers in dogs.

Finasteride affects young male rats' brain function and behavior negatively.

Men need higher doses of botox than women and a different approach for facial aesthetics due to their unique facial features and muscle mass.

Male pattern baldness may be caused by factors like poor blood circulation, scalp tension, stress, and hormonal imbalances, but the exact causes are still unclear.

Hyperadrenocorticism in ferrets is linked to neutering and indoor housing, and is best treated with surgery and a deslorelin implant.

Iron levels are linked to insulin resistance in men and postmenopausal women, but not in premenopausal women.

Finasteride treatment changes Cx43 in rat testes, possibly causing fertility issues.

Cancer treatments can cause skin-related side effects that may affect patient quality of life and require changes in treatment.

Hair loss in Androgenetic alopecia (AGA) is due to altered cell sensitivity to hormones, not increased hormone levels. Hair growth periods shorten over time, causing hair to become thinner and shorter. This is linked to miscommunication between cell pathways in hair follicles. There's also a change in gene expression related to blood vessels and cell growth in balding hair follicles. The exact molecular causes of AGA are still unclear.

The workshop highlighted the genetic links and psychological impacts of hair loss and skin disorders.

PCOS is a complex disorder in women that can lead to various health risks and requires personalized treatment.

Insulin resistance is a key factor in polycystic ovary syndrome, but genetics may also contribute.

Humans lost body hair relatively recently in evolution.

The conclusion is that PCOS is caused by ovarian sensitivity to hormones and disrupted hormone control, possibly due to ovarian factors, and more research is needed.

Recombinant cytokine therapy can cause skin reactions ranging from mild to severe.

Tissue-resident memory T cells can protect against infections and cancer but may also contribute to autoimmune diseases.

Nude mice with grafted human skin developed scars similar to human hypertrophic scars.

The vitamin D receptor is essential for skin stem cells to grow, move, and become different cell types needed for skin healing.

Mutations in the PTPRQ gene cause significant balance issues in mice due to hair bundle defects in the inner ear.

Epidermal keratinocytes start wound healing and inflammation after schistosome infection.

Facial aging is caused by natural processes and external factors, and can be managed with preventative measures and a variety of treatments tailored to individual needs.

Human hair has more unsaturated fats inside than on the surface, and certain lipids may help bind the outer and inner layers together.

Bioluminescence imaging can track hair follicle cells and help study hair regrowth.

The document concludes that hormonal biomarkers are key for diagnosing hyperandrogenemia in women and hypogonadism in men.

Dutasteride may help reduce brain plaque linked to Alzheimer's by affecting cell energy structures and waste removal.