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Uncombable hair syndrome causes frizzy hair and can affect the nervous system, eyes, and ears, often co-occurring with other hair, skin, nail, and teeth conditions, and is linked to three specific gene mutations.

Testosterone, progesterone, and levonorgestrel change enzyme levels related to fat production in hamster skin, which could affect skin oil and acne.

The document concludes that targeting the androgen receptor may be a promising breast cancer treatment, especially for certain types.

Certain changes to the B-ring of androgen receptor ligands can increase their effectiveness for potential treatments of muscle and bone conditions.

The conclusion is that skin and hair patterns are formed by a mix of cell activities, molecular signals, and environmental factors.

The same hormone can affect gene expression differently in various tissues, which could lead to new treatments for conditions like hair loss.

Estrogens are key for bone growth spurts in both boys and girls and affect growth into adulthood.

Genetic variants at 20p11 increase baldness risk in Chinese Han people.

Analog 23 is a promising compound for prostate cancer treatment.

Testosterone can both promote hair growth and cause baldness by affecting hair growth signals.

The mouse model suggests male pattern baldness may be due to an enzyme increasing DHT and higher androgen receptor levels in hair follicles.

Common latent viruses might contribute to male-pattern baldness by disrupting cell processes that normally suppress hair loss-related proteins.

A specific gene variant is linked to heart disease, increased heart muscle, curly hair, and thick skin on palms and soles.

Androgens may cause hair loss by increasing TGF-beta1 from scalp cells, which inhibits hair cell growth.

Androgens can help prevent memory problems caused by apoE4.

CDP is crucial for lung and hair follicle cell development.

Matriptase is crucial for keeping epithelial tissues healthy and functioning properly.

Male pattern baldness involves hormones and cell signals affecting hair growth.

The research found that Atypical Progeroid Syndrome has unique symptoms and is not caused by the buildup of a certain mutant protein.

People with PCOS, especially if obese, often have NAFLD, linked to obesity, insulin resistance, and high androgen levels.

Lampreys have a functional vitamin D receptor that may help detoxify harmful substances.

The document concludes that early diagnosis and treatment of Congenital Adrenal Hyperplasia are crucial for preventing serious health issues and improving patient outcomes.

Hidradenitis suppurativa is caused by genetic factors, inflammation, bacteria, hormones, and lifestyle factors like obesity and smoking.

Cells in hair follicles help create fat cells in the skin by releasing a protein called Sonic Hedgehog.

Sphynx cats are hairless and Devon Rex cats have curly hair due to specific genetic mutations.

Stress increases certain chemicals in the skin and nerves, which might worsen skin conditions.

Hair sensitivity to androgens is partly controlled by specific enzyme expressions in different hair areas.

The review recommends hormone replacement therapy for women with premature ovarian insufficiency to manage symptoms and protect health, with specific approaches for different groups.

The article explains the genetic causes and symptoms of various hair disorders and highlights the need for more research to find treatments.

Nonclassic adrenal hyperplasia is a genetic condition that can cause early puberty and fertility problems, treated with specific steroids.