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Damaging mitochondrial DNA in mice speeds up aging due to increased reactive oxygen species, not through the p53/p21 pathway.

Injury changes how hair follicle stem cells behave, depending on the hair growth stage.

Stress stops hair growth in mice by causing early hair growth phase end and harmful inflammation through a specific nerve-related pathway.

Prolactin affects hair growth cycles and can cause early hair follicle regression.

Stress increases certain chemicals in the skin and nerves, which might worsen skin conditions.

Keratin 17 is modified by RSK1 in response to growth and stress, affecting skin growth and stress response.

XMU-MP-1 stops cell growth in a human mini-organ and reduces the effectiveness of the chemotherapy drug paclitaxel.

Type 2 diabetes slows down skin and hair renewal by blocking important stem cell activation in mice.

Changing CDK4 levels affects the number of stem cells in mouse hair follicles.

Auto-antibody testing is a useful but not definitive tool in diagnosing interstitial lung diseases, and using a specific algorithm could make testing more cost-effective.

Smad2/3-dependent TGF-β signaling increases during wound healing.

Younger mice's hair-follicle stem cells are better at turning into heart cells than older mice's.

High levels of ERK activity are key for tissue regeneration in spiny mice, and activating ERK can potentially redirect scar-forming healing towards regenerative healing in mammals.

A form of vitamin E promotes hair growth by activating a specific skin pathway.

Normal human melanocytes can avoid cell death through multiple pathways.

Blocking the protein CXCL12 with a specific antibody can increase hair growth in common hair loss conditions.

Hyaluronate fragments can help reverse skin thinning by working with the CD44 receptor.

Noggin gene inactivation causes skeletal defects in mice, varying by genetic background.

Sex and sex hormones can affect brain inflammation in Parkinson's disease, with male mice being more affected and female mice showing a protective effect.

Stress in hair follicle stem cells causes inflammation in a chronic skin condition through a specific immune response pathway.

Sheep hair follicle cells can grow a lot but need the dermal papilla to do so.

Hydroxyl radicals cause hair follicle cell death during chemotherapy by reducing Bcl-2 protein levels.

Circular RNA ERCC6 helps activate stem cells important for cashmere goat hair growth by interacting with specific molecules in an m6A modification-dependent way.

A woman developed severe skin damage after using a blood thinner called low-molecular-weight heparin.

Most people with scarring and nonscarring hair loss show similar D2-40 levels, but some with scarring hair loss have higher levels.

The enzyme steroid sulfatase is linked to breast cancer and other conditions, and inhibitors are being developed for treatment.

Chemotherapy causes hair loss by damaging hair follicles and stem cells, with more research needed for prevention and treatment.

Hair follicle development and microbes help regulatory T cells gather in newborn skin.

5α-reductase is essential for male sexual development and its inhibitors have potential in treating various conditions related to hormone action.

S100A3 protein is crucial for hair shaft formation in mice.