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Research 421–450 of 1000+
- Oral spironolactone therapy for female patients with acne, hirsutism or androgenic alopecia
- Follicular delivery of spironolactone via nanostructured lipid carriers for management of alopecia
- Androgen excess: Investigations and management
- Polycystic Ovary Syndrome: A Comprehensive Review of Pathogenesis, Management, and Drug Repurposing
- Treatment Options for Hirsutism: A Systematic Review and Network Meta-Analysis
- Dermatology of androgen-related disorders
- Medical treatment of hirsutism
- Androgen metabolism in hirsute patients treated with cyproterone acetate
- New Therapeutic Approach to the Hirsute Patient
- Treatment of hirsutism and acne in hyperandrogenism
- Use of ethinylestradiol/drospirenone combination in patients with the polycystic ovary syndrome
- Hormonal Treatment Strategies Tailored to Non-Binary Transgender Individuals
- Nutrition Strategy and Life Style in Polycystic Ovary Syndrome—Narrative Review
- Hidradenitis suppurativa: an-androgen-dependent disorder
- Hormonal treatment of acne vulgaris: an update
- Steroid 5α-Reductase Inhibitors
- Evidence-based approach to cutaneous hyperandrogenism in women
- Recent advances in oxasteroids chemistry
- Androgens Affect the Activity of Human Sebocytes in Culture in a Manner Dependent on the Localization of the Sebaceous Glands and Their Effect Is Antagonized by Spironolactone
- Spironolactone Therapy for Hirsutism in a Hyperandrogenic Woman
- Non polycystic ovary syndrome–related endocrine disorders associated with hirsutism
- Consecutive Cyproterone Acetate and Estradiol Treatment in Late-Pubertal Transgender Female Adolescents
- In vitro bioassays for androgens and their diagnostic applications
- A Clinical Approach to Severe Insulin Resistance
- Cardiovascular risk factors and events in women with androgen excess
- Androgens and Anti-androgen Therapy
- Endocrine evaluation of hirsutism
- Becker's Nevus with Ipsilateral Breast Hypoplasia: Improvement with Spironolactone
- Flutamide in the treatment of hirsutism
- Contribution of androgens to chronic allograft nephropathy is mediated by dihydrotestosterone