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Regenerative cosmetics can improve skin and hair by reducing wrinkles, healing wounds, and promoting hair growth.

The report concludes that PCOS is mainly a condition of excess male hormones and its definition may change as new information is discovered.

People with hair loss have more androgen receptors and enzymes in certain follicles, with men and women showing different patterns.

The Endocrine Society advised against routine testosterone therapy for women, citing a need for more research on long-term safety and a clear definition of androgen deficiency.

The guidelines advise against using testosterone and DHEA in women for most conditions due to safety and effectiveness concerns, but suggest considering testosterone for postmenopausal women with low sexual desire.

Mutations at Val-889 and Arg-752 disrupt key interactions in the androgen receptor, affecting its function.

ERG increases SOX9, promoting prostate cancer growth and invasion.

Genetic variation in the androgen receptor gene mainly causes early-onset hair loss, with maternal inheritance playing a key role.

Five new mutations in the androgen receptor gene were found, helping to understand androgen insensitivity syndrome better.

Estrogen Receptor ß (ERß) is the main hormone controller in human skin and hair follicles, not Estrogen Receptor α (ERα) or the Androgen Receptor (AR).

Too much male hormone is the main cause of Polycystic Ovary Syndrome.

Testosterone replacement may help some postmenopausal women with symptoms like fatigue and low libido, but more research is needed to fully understand its effects.

E211 G>A gene linked to lower risk of severe prostate cancer and hair loss.

Genetic differences may affect COVID-19 deaths; anti-androgens could be potential treatment.

Women with PCOS have higher androgen levels that decrease with age but are still higher than in women without PCOS.

Androgen receptors are key for development and health, affecting conditions like prostate cancer and male pattern baldness.

Hair sensitivity to androgens is partly controlled by specific enzyme expressions in different hair areas.

Androgens are important for normal ovarian function and estrogen production, but may not be the main cause of follicle death.

The balance between androgen receptor and p53 is crucial for sebaceous gland differentiation.

Testosterone replacement can help women with low libido and mood, but they need to have enough estrogen first to avoid side effects.

Steroidogenesis inhibitors change but don't stop androgen production in prostate cancer.

The length of the CAG repeat in the androgen receptor gene affects the risk and progression of prostate cancer, BPH, infertility, and undermasculinized genitalia.

Use "female pattern hair loss" term, assess androgen excess, treat with minoxidil and other medications if needed.

Testosterone helps beard and axillary hair cells grow by releasing growth factors from dermal papilla cells.

Androgen receptor activity blocks Wnt/β-catenin signaling, affecting hair growth and skin cell balance.

Male genital development is driven by androgen signaling and understanding it could help address congenital anomalies.

Longer CAG repeat lengths in the androgen receptor gene are linked to higher testosterone levels in women with PCOS.

Androgen is important in controlling stem cell differentiation, reducing fat development, and increasing lean mass.

Shorter CAG repeats in a specific gene may increase male hormone activity and symptoms like acne and excess hair in women with PCOS.

Testing for CAG repeat polymorphism in the androgen receptor gene is not currently recommended for managing hypogonadism.