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Research 181–210 of 444
- Polycystic Ovary Syndrome
- Delayed Puberty
- Modelling androgen synthesis and action during human sexual differentiation
- Safety profile of ipilimumab in elderly patients: a disproportionate analysis based on the FDA Adverse Event Reporting System database
- Secondary Adrenal Insufficiency : A Case Report
- Genetics and Pathophysiology of Congenital Adrenal Hyperplasia
- Primary Generalized Glucocorticoid Resistance or Chrousos Syndrome: Allostasis Through a Mutated Glucocorticoid Receptor
- Diagnostic approach to low‐renin hypertension
- Polycystic Ovary Syndrome, Insulin Resistance, and Obesity: Navigating the Pathophysiologic Labyrinth
- 46,XY DSD due to impaired androgen production
- Endocrine-skin interactions
- Alternative treatment of polycystic ovary syndrome: pre-clinical and clinical basis for using plant-based drugs
- The rationale for physiological testosterone replacement in women
- Whole Genome Scan and Selection Signatures for Climate Adaption in Yanbian Cattle
- Polycystic ovary syndrome
- The role of androgens in hormone replacement therapy
- The Case of the Elusive Androgen
- Hair Cortisol: A Biomarker of Chronic Stress in Animals and its Association with Reproduction
- Contaminants of emerging concern in the fetal environment
- Long-term physiological trends and their drivers: linking hair hormone concentrations with telemetry data in GPS-collared Serengeti wildebeest
- Unravelling the link between insulin resistance and androgen excess
- ACNE
- Cardiovascular risk factors and events in women with androgen excess
- An Update on Drug-Induced Pigmentation
- Hyperandrogenism in polycystic ovary syndrome and adrenal hyperplasia: finding differences to make a specific diagnosis
- Men??s Health
- Pharmacology of antiandrogens
- Endocrinological evaluation and hormonal therapy for women with difficult acne
- Glucocorticoids Influencing Wnt/β-Catenin Pathway; Multiple Sites, Heterogeneous Effects
- An N-Ethyl-N-Nitrosourea Induced Corticotropin-Releasing Hormone Promoter Mutation Provides a Mouse Model for Endogenous Glucocorticoid Excess