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The document concludes that assessing hair follicle damage due to cyclophosphamide in mice involves analyzing structural changes and suggests a scoring system for standardized evaluation.

Restoring hair bulb immune privilege is crucial for managing alopecia areata.

Grafted rodent and human cells can regenerate hair follicles, but efficiency decreases with age.

Notch/CSL signaling controls hair follicle differentiation through Wnt5a and FoxN1.

Hair follicle regeneration needs special conditions and young cells.

Follistatin helps hair growth and cycling, while activin prevents it.

Stress hormone corticosterone blocks a growth factor to slow down hair stem cell activity and hair growth.

Muscles and nerves that cause goosebumps also help control hair growth.

PRP and HF-MSCs treatment improves hair growth, thickness, and density in androgenetic alopecia.

An imbalance between certain immune cells is linked to a chronic skin condition and may be influenced by obesity, smoking, and autoimmune issues.

The document concludes that for hair and feather growth, it's better to target the environment around stem cells than the cells themselves.

The study showed that hair follicle stem cells can maintain and organize themselves in a lab setting, keeping their ability to renew and form hair and skin.

miR-22, a type of microRNA, controls hair growth and its overproduction can cause hair loss, while its absence can speed up hair growth.

The document concludes that mouse models are crucial for studying hair biology and that all mutant mice may have hair growth abnormalities that require detailed analysis to identify.

Bcl-2 overexpression protects against UVB damage but worsens hair loss from chemotherapy.

Lymphatic vessels are essential for hair follicle growth and skin regeneration.

Disrupting Smad4 in mouse skin causes early hair follicle stem cell activity that leads to their eventual depletion.

Macrophages help hair growth after injury through CX3CR1 and TGF-β1.

Cells from a skin condition can create new hair follicles and similar growths in mice, and a specific treatment can reduce these effects.

Melatonin helps Cashmere goats grow more hair by affecting certain genes and cell pathways.

Smad1 and Smad5 are essential for hair follicle development and stem cell sleepiness.

Gata6 is important for protecting hair growth cells from DNA damage and keeping normal hair growth.

Loss of Fz6 disrupts hair follicle and associated structures' orientation.

Dogs could be good models for studying human hair growth and hair loss.

Endoglin is crucial for proper hair growth cycles and stem cell activation in mice.

Cholesterol balance is important for hair health, and problems with it can lead to hair loss conditions.

Cashmere and milk goats have different hair growth cycles and gene expressions, which could help improve wool production.

Blocking Wnt/β-catenin signaling with EGF receptor is necessary for proper hair growth.

Changing light exposure can affect hair growth timing in goats, possibly due to a key gene, CSDC2.

NF-κB is crucial for different stages and types of hair growth in mice.