Search

everythinglearnresearchcommunity

for

Search:↓

Increased Toll-like receptors in blood cells may contribute to alopecia areata and could be a target for new treatments.

SHISA6 helps maintain certain stem cells in mouse testes by blocking signals that would otherwise cause them to differentiate.

Collagen VII helps skin heal and stay strong, sirolimus may lower skin cancer risk in kidney transplant patients, high-molecular-mass hyaluronan helps naked mole rats resist cancer, dermal γδ T cells aid in hair growth in rodents, and overexpression of IL-33 in mouse skin causes itchiness, offering a model for studying allergic inflammation treatments.

TGF-β is crucial for tissue repair and can cause diseases if not properly regulated.

Grouping certain skin cells together activates a growth pathway that helps create new hair follicles.

The symposium concluded that a specific lipid might cause hair loss, inflammation is a key factor, and stem cells in bald scalps aren't working right, suggesting new treatment targets.

Genetic mutation and carcinogen treatment are both needed for skin cancer to develop in these specific mice.

TLR3 signaling enhances the immunosuppressive properties of human periodontal ligament stem cells.

Increasing Treg cells in the skin does not cure hair loss from alopecia areata in mice.

ILC1 cells contribute to hair loss in alopecia areata.

Increasing the levels of a protein called FGF9 can promote hair growth, but humans may not respond the same way due to a lack of certain cells.

Immune cells are crucial for hair growth and preventing hair loss.

Alopecia areata can be reversed by JAK inhibitors, promoting hair regrowth.

The review suggests that other immune cells besides CD8+ T cells may contribute to alopecia areata and that targeting regulatory cell defects could improve treatment.

Mice with human fetal thymic tissue and stem cells developed symptoms similar to chronic graft-versus-host disease.

Special immune cells called Tregs can help prevent lung scarring by blocking a specific growth factor.

New hair can grow from large wounds in mice, but less so as they age, involving reprogramming of skin cells and specific molecular pathways.

Deleting the p63 gene in certain cells causes problems in thymus development and severe hair loss in mice.

Deleting the BRD4 protein in certain skin cells causes hair loss and skin inflammation.

Jagged-1 in skin Tregs is crucial for timely wound healing by recruiting specific immune cells.

The research suggests that immune cells and a specific type of cell death called ferroptosis are involved in Frontal fibrosis alopecia.

A protein called EGFR protects hair follicle stem cells, and when it's disrupted, hair follicles can be damaged, but blocking certain pathways can restore hair growth.

The FOXN1 gene is crucial for developing immune cells and preventing immune disorders.

Immune cells are essential for skin regeneration using biomaterial scaffolds.

Immune cells affect hair growth and could lead to new hair loss treatments.

RANKL improves the immune response against herpes simplex virus by enhancing T cell activation and could help develop better treatments or vaccines.

Th22 cells are essential for Tβ15-induced hair growth in mice.

Mice treatments didn't grow hair, a patient treatment may affect immune response, and people with hair loss often feel anxious or depressed.

LL-37 helps stem cells grow and move, aiding tissue regeneration and hair growth.

External factors can cause skin cancer cells that usually don't spread to grow and form tumors in mice.