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Research 211–240 of 1000+
- Clinical use of 5α-reductase inhibitors
- The Polycystic Ovary Syndrome - Challenges and Opportunities in Adolescent Medicine
- Adrenal causes of endocrine hypertension in childhood or adolescence
- Acne-Associated Syndromes
- Relationships between cerebrospinal fluid GABAergic neurosteroid levels and symptom severity in men with PTSD
- Targets to treat androgen excess in polycystic ovary syndrome
- Hyperadrenocorticism in Ferrets
- Lack of association between clinical and biochemical hyperandrogenism in patients with polycystic ovary syndrome
- In vivo and in vitro exposure to free fatty acids induce increased androgen production in animal models: a potential pathogenic factor of the polycystic ovary syndrome
- Polycystic ovary syndrome
- Melatonin membrane receptors in peripheral tissues: Distribution and functions
- Is dehydroepiandrosterone a hormone?
- Skin melanocytes: biology and development
- The cutaneous serotoninergic/melatoninergic system: securing a place under the sun
- Polycystic Ovary Syndrome, Insulin Resistance, and Obesity: Navigating the Pathophysiologic Labyrinth
- Androgen Receptor Antagonists (Antiandrogens) Structure-Activity Relationships
- Sex differences in the hypothalamic–pituitary–adrenal axis’ response to stress: an important role for gonadal hormones
- The pathophysiology of polycystic ovary syndrome: trying to understand PCOS and its endocrinology
- Neurogenic Inflammation in Stress-Induced Termination of Murine Hair Growth Is Promoted by Nerve Growth Factor
- Androgens and androgen receptor action in skin and hair follicles
- Pharmacology of antiandrogens
- Hormonal Therapy for Acne
- <i>Polycystic Ovary Syndrome in Adolescence</i>
- Neurosteroids, neuroactive steroids, and symptoms of affective disorders
- PAK4 signaling in health and disease: defining the PAK4–CREB axis
- Hair Loss in Women
- MENSTRUAL DISORDERS IN ADOLESCENTS
- 9 Pilosebaceous physiology in relation to hirsutism and acne
- Menstrual Irregularity in Women with Acromegaly
- Recent advances in the molecular mechanisms determining tissue sensitivity to glucocorticoids: novel mutations, circadian rhythm and ligand-induced repression of the human glucocorticoid receptor