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The condition called 'acute diffuse and total alopecia of the female scalp' is actually a known condition named alopecia areata incognita.

ULBP3 could be a marker for diagnosing alopecia areata incognita and may be linked to its cause and development.

Understanding the genetics of alopecia areata could lead to better treatments.

Higher CD70 and CD27 gene expression in alopecia areata lesions predicts disease severity and activity.

Human Beta Defensin 1 levels do not predict the risk or severity of Alopecia Areata.

New treatments for hair growth and psoriasis may be possible, and gene differences could affect baldness and the severity of skin conditions.

R-spondin2 may help treat hair loss, gene differences could explain baldness, a peptide's regulation is linked to psoriasis, B-defensin gene copies may affect a skin condition's risk and severity, and potential markers and targets for alopecia areata were identified.

Mast cells play a significant role in hair loss conditions like male pattern hair loss and alopecia areata.

Common baldness is a hereditary condition that can be treated with medications or surgery to prevent progression and improve self-esteem.

Some treatments for autoimmune hair loss work, but JAK inhibitors like tofacitinib are promising for regrowth.

People with alopecia areata often have thyroid autoimmunity.

Different genes are linked to various types of hair loss.

The conclusion is that acne, alopecia, and hyperhidrosis are common skin issues with various treatments available, and accurate diagnosis is key for effective management.

No CD44 in alopecia areata, present in normal and androgenetic alopecia.

Baricitinib is a new, effective treatment for advanced alopecia areata but is costly and has risks.

A specific gene variant (rs4833095) is linked to a higher risk of alopecia areata in Koreans.

Alopecia Areata is a hair loss condition affecting all genders, often linked to other diseases, with treatments available but varying success rates.

Azelaic acid may help treat acne by killing bacteria, reducing inflammation, and preventing clogged pores.

Better models and evaluation methods for alopecia areata are needed.

Alopecia areata is likely caused by a combination of genetic factors and immune system dysfunction, and may represent different diseases with various causes.

Certain proteins, caspases-1 and -11, are important in the early development of skin inflammation in mice.

Scalp cooling is the most effective FDA-approved method to prevent chemotherapy-induced hair loss, but more research is needed for other treatments.

Alopecia Areata significantly lowers quality of life and current treatments are inadequate, highlighting a need for better therapies and standardized treatment protocols.

The model suggests that scalp tension could lead to hair loss, with factors like blood vessel hardening, enlarged oil glands, and poor microcirculation also playing a role. It also hints at a possible link between skull shape and baldness pattern.

Trichilemmal carcinoma shares genetic traits with other skin cancers, suggesting similar treatment options.

Trichilemmal carcinoma shares genetic traits with other skin cancers, suggesting similar treatment options.

A man with vitiligo and alopecia saw quick skin and hair improvement with ruxolitinib, but skin color gains were lost after stopping treatment.

Kennedy's disease leads to muscle weakness without a cure, but exercise and managing symptoms may help patients live a normal lifespan.

The cause of alopecia areata is likely a mix of genetics, immune system issues, and environmental factors, with more research needed to understand it fully.

Damaged hair follicles make mice more prone to skin inflammation and skin cancer after UV exposure.