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Skin development in mammals is controlled by key proteins and signals from underlying cells, involving stem cells for maintenance and repair.

Mouse amnion can turn into skin and hair follicles with help from certain cells and factors.

ΔNp63α stops TAp73β from working in skin cancer by blocking its access to specific genes, not by directly interacting with it.

The protein Par3 is crucial for healthy skin, affecting the skin barrier, cell differentiation, and stem cell maintenance.

Intu gene is crucial for hair follicle formation by helping keratinocytes differentiate through primary cilia.

Removing centrosomes from skin cells leads to thinner skin and stops hair growth, but does not greatly affect skin cell differentiation.

Adam10 enzyme is crucial for healthy skin and proper Notch signaling.

Tiny needles with valproic acid can effectively regrow hair.

Skin tumor regression is helped by retinoic acid signaling blocking Wnt signaling.

Heparan sulfate is important for hair growth, preventing new hair formation in mature skin, and controlling oil gland development.

Claudin-1 is important for the barrier function and growth of hair.

Cells from a skin condition can create new hair follicles and similar growths in mice, and a specific treatment can reduce these effects.

Hair follicle regeneration may slow tumor growth.

Skin abnormalities can indicate immunodeficiency due to shared origins with the immune system.

Hair growth environment recreated with challenges; stem cells make successful skin organoids.

Stem cells are crucial for skin repair and new treatments for chronic wounds.

Overexpression of activin A in mice skin causes skin thickening, fibrosis, and improved wound healing.

Tight junctions are key for skin protection and controlling what gets absorbed or passes through the skin.

Too much Smad7 changes skin and hair development by breaking down a protein called β-catenin, leading to more oil glands and fewer hair follicles.

Different scaffold patterns improve wound healing and immune response in mouse skin, with aligned patterns being particularly effective.

The extracellular matrix is crucial for controlling skin stem cell behavior and health.

Mice genetically modified to produce more CD109 in their skin had less inflammation and better healing with less scarring.

Telomere damage affects skin and hair follicle stem cells by messing up important growth signals.

HPV genes and estradiol increase a cancer-related signaling pathway, which may be targeted for cervical cancer treatment.

Newly found stem cells in horse hooves show promise for treating a hoof disease called laminitis.

Clim proteins are essential for maintaining healthy corneas and hair follicles.

Cell polarity signaling controls tissue mechanics and cell fate, with complex interactions and varying pathways across species.

Mitochondrial reactive oxygen species are essential for skin and hair development.

Epidermal stem cells help heal severe skin wounds and have potential for medical treatments.

Lack of Ctip2 in skin cells delays wound healing and disrupts hair follicle stem cell markers in mice.