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The research found a way to identify and study skin cells with stem cell traits, revealing they behave differently in culture and questioning current stemness assessment methods.

The research found ways to activate melanocyte stem cells for potential treatment of skin depigmentation conditions.

Hair follicle stem cells use specific chromatin changes to control their growth and differentiation.

Aging mice have slower hair regeneration due to changes in signal balance, but the environment, not stem cell loss, controls this, suggesting treatments could focus on environmental factors.

Keratin 17 is modified by RSK1 in response to growth and stress, affecting skin growth and stress response.

Transplanted hair follicles can change and adapt to new areas of the body, with the immune system possibly playing a role in this adjustment.

Adipocytes can change into fibroblast-like cells to help with wound healing.

EdnrB signaling helps melanocyte stem cells regenerate and could be targeted to treat pigmentation issues.

The vitamin D receptor is essential for skin stem cells to grow, move, and become different cell types needed for skin healing.

Activating Notch in adult skin causes T cells and neural crest cells to gather, leading to skin issues.

Hair and skin can regenerate without bulge stem cells due to other compensating cells.

The research reveals how early embryonic mouse skin develops from simple to complex structures, identifying various cell types and their roles in this process.

TLR2 is important for hair growth and can be targeted to treat hair loss.

Older people's sweat glands are less effective at helping skin wounds heal due to weaker cell connections.

Dermoscopy is a useful tool for identifying features of skin conditions, but more research is needed to define its role in dermatology.

The nude gene causes skin cell overgrowth and improper development, leading to hair and urinary issues.

Steven Kossard classified lymphocyte-related hair loss into four patterns, each linked to different types of baldness.

GRK2 is essential for healthy hair follicle function, and its absence can lead to hair loss and cysts.

Disrupted cholesterol production impairs hair follicle stem cells, leading to hair loss.

Skin lesions in Carney complex are likely caused by a specific group of skin cells that promote pigment production due to a genetic mutation.

Too much Smad7 changes skin and hair development by breaking down a protein called β-catenin, leading to more oil glands and fewer hair follicles.

The "flying-wings" scalp flap technique is a simple, safe, and effective way to reconstruct large areas of scalp loss in children.

Dermal papillae cells, important for hair growth, come from multiple cell lines and can be formed by skin cells, regardless of their origin or hair cycle phase. These cells rarely divide, but their ability to shape tissue may contribute to their efficiency in inducing hair growth.

A mouse gene mutation increases the risk of skin cancer.

Trichoscopy is useful for diagnosing hair and scalp disorders in people with darker skin.

YAP and TAZ are crucial for skin cell growth and repair.

A reliable system was developed to distinguish hair growth stages, aiding in identifying hair growth promoters or inhibitors.

Researchers found genes linked to hair growth cycles in Inner Mongolia cashmere goats, which could help understand and treat hair loss.

A new treatment using nanoparticles can effectively prevent and reduce hair loss caused by chemotherapy.

Removing Mediator 1 from certain mouse cells causes teeth to grow hair instead of enamel.