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Vitamin D and estrogen may help protect heart and kidney health, and maintaining sufficient vitamin D levels could be especially beneficial for African Americans, postmenopausal women, and people with chronic kidney disease.

Two rare genetic diseases cause severe rickets in children due to defects in vitamin D metabolism.

Vitamin D and calcium are important for skin stem cell function and wound healing.

Human skin acts like a hormone-producing organ, making and managing various hormones important for skin and hair health.

Aging reduces skin stem cell function, leading to changes like hair loss and slower wound healing.

The R343H mutation in the VDR gene causes vitamin D-resistant rickets with alopecia by impairing specific gene activity.

Some vitamin D analogs can thicken skin and reduce pore size like a common acne treatment, with one analog also affecting skin growth factors.

Melasma is a skin condition linked to female hormones, genetics, UV exposure, and certain medications, but not to pituitary, adrenal, or thyroid diseases.

Vitamin D3 may protect rat hair follicles from radiation damage.

Vitamin D Receptor is crucial for normal skin and hair growth.

Vitamin D is crucial for bone health and preventing serious diseases.

Child with rickets improved with a specific vitamin D therapy, but alopecia did not change.

A child with rickets and hair loss might have a rare type of rickets that doesn't improve much with usual vitamin D and calcium treatment.

A boy with severe Vitamin D-resistant rickets did not respond to treatment and lacked a common symptom, suggesting a need for alternative treatments.

The enzyme 25 Hydroxyvitamin D 1 α-Hydroxylase is essential for healthy skin and recovery after skin damage.

Vitamin D has potential benefits for cancer prevention, heart health, diabetes, obesity, muscle function, skin health, and immune function, but clinical results are mixed and more research is needed.

Some people have genetic mutations that make them resistant to vitamin D, leading to rickets even with enough vitamin D intake.

Kids with alopecia areata had lower vitamin D levels than healthy kids.

Mice without Vitamin D receptors have hair growth problems because of issues in the hedgehog signaling pathway.

Mice without the vitamin D receptor gene lose hair due to disrupted hair follicle cycles.

Low vitamin D levels are common in people with Alopecia Areata.

Vitamin D might be involved in the development of alopecia areata and could help in its treatment.

Certain natural products may help stimulate hair growth by affecting stem cell activity in the scalp.

Gene therapy helped rats with a specific type of rickets grow hair without severe inflammation.

As skin cells mature, vitamin D receptor levels decrease while retinoid X receptor α levels increase.

Combination therapies might work better for some vitiligo patients, but results vary.

The document concludes that blocking the internal pathways that create androgens might help treat cancers that depend on sex hormones.

Vitamin D3 affects cell differentiation in specific skin areas.

Vitamin D is important for skin health and can help treat psoriasis, atopic dermatitis, and vitiligo.

A 3-year-old with vitamin D-resistant rickets and severe hair loss died despite treatment, highlighting the need for improved management of the condition.