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Three siblings with a genetic form of rickets showed different symptoms of the disease.

The research showed that Vitamin D and its receptor are important for healthy bones and normal hair and skin in rats.

1α,25-dihydroxyvitamin D3 directly affects cartilage growth and development.

Removing vitamin D and calcium receptors in mice skin cells slows down skin wound healing.

Vitamin D has potential benefits for cancer prevention, heart health, diabetes, obesity, muscle function, skin health, and immune function, but clinical results are mixed and more research is needed.

The vitamin D receptor is involved in multiple body functions beyond calcium regulation, including immune response and rapid reactions not related to gene activity.

The mTOR signaling pathway is crucial for hair health and targeting it may lead to new hair loss treatments.

The vitamin D receptor has many roles in the body beyond managing calcium, affecting the immune system, hair growth, muscles, fat, bone marrow, and cancer cells.

Gene therapy helped rats with a specific type of rickets grow hair without severe inflammation.

The study found that certain mutations in the vitamin D receptor can cause rickets and potentially affect hair growth.

Vitamin D helps skin stem cells heal wounds by working with a key skin protein.

Mutations in the vitamin D receptor cause hereditary vitamin D-resistant rickets, leading to poor bone health and requiring high calcium doses for treatment.

Vitamin A toxicity likely caused the patient's high calcium levels.

Vitamin D receptor is important for regulating hair growth and wound healing in mice.

DHT inhibition may increase spinal bone growth in ankylosing spondylitis.

Low Vitamin D receptor levels in breast cancer are linked to worse outcomes and more bone metastases, and could be a marker for prognosis.

Vitamin D3 affects cell differentiation in specific skin areas.

Vitamin D receptor interacts with certain dietary components to help prevent diseases and regulate hair growth.

Vitamin D and its receptor regulate skin functions like cell growth, immunity, hair cycle, and tumor prevention.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.

A 3-year-old with vitamin D-resistant rickets and severe hair loss died despite treatment, highlighting the need for improved management of the condition.

Vitamin D is important for skin health and may affect conditions like psoriasis and hair loss, but more research is needed to understand its role fully.

The enzyme 25 Hydroxyvitamin D 1 α-Hydroxylase is essential for healthy skin and recovery after skin damage.

A genetic defect in vitamin D receptors causes severe rickets and hair loss in children, but some heal as they age.

Vitamin D3 is important for bone health and may help treat various diseases beyond bone-related conditions.

Patients with this syndrome can have different responses and worsening resistance to treatment over time.

A girl with rickets and hair loss was found to have a rare condition where her body didn't respond to vitamin D treatment.

GLP-1 receptor agonists, like Dulaglutide, Liraglutide, and Semaglutide, have potential benefits beyond the pancreas, including neuroprotection, pain suppression, cardiovascular protection, obesity management, and cancer treatment, but there are concerns about pancreatitis and pancreatic cancer risks.

Minoxidil and pyrithione zinc combo most effectively increases hair density.

A boy with severe Vitamin D-resistant rickets did not respond to treatment and lacked a common symptom, suggesting a need for alternative treatments.