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A genetic defect in vitamin D receptors causes severe rickets and hair loss in children, but some heal as they age.

HVDRR is caused by VDR gene mutations, leading to vitamin D resistance, treatable with high calcium doses, but alopecia remains permanent.

A girl with rickets and hair loss was found to have a rare condition where her body didn't respond to vitamin D treatment.

A vitamin D receptor mutation causes rickets and affects immune responses.

Three siblings with a genetic form of rickets showed different symptoms of the disease.

Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.

A boy with severe Vitamin D-resistant rickets did not respond to treatment and lacked a common symptom, suggesting a need for alternative treatments.

Mice without the vitamin D receptor have bone issues and other health problems, suggesting vitamin D is important for preventing various diseases in humans.

A child with rickets and hair loss might have a rare type of rickets that doesn't improve much with usual vitamin D and calcium treatment.

The R343H mutation in the VDR gene causes vitamin D-resistant rickets with alopecia by impairing specific gene activity.

Vitamin D and its receptor regulate skin functions like cell growth, immunity, hair cycle, and tumor prevention.

Some people have genetic mutations that make them resistant to vitamin D, leading to rickets even with enough vitamin D intake.

Vitamin D3 analogs can promote hair growth in mice genetically prone to hair loss.

Two rare genetic diseases cause severe rickets in children due to defects in vitamin D metabolism.

Two different mutations in the vitamin D receptor gene cause different symptoms and responses to treatment in Lebanese patients with hereditary rickets.

Vitamin D and its receptor are essential for hair growth, cell regulation, immune function, and heart health.

Systemic diseases can cause hair loss, which is often reversible with treatment.

Mutations in the vitamin D receptor cause hereditary vitamin D-resistant rickets, leading to poor bone health and requiring high calcium doses for treatment.

A Chinese family had a child with a specific gene mutation causing vitamin D-resistant rickets, but the child improved with calcium and low-dose calcitriol.

Intravenous calcium therapy is a safe and effective treatment for a rare type of rickets.

Taking vitamin D might improve life for MS patients and reduce skin side effects from alemtuzumab treatment.

People with alopecia areata often have lower levels of vitamin D, zinc, and folate, but more research is needed to understand if supplements can help treat it.

Some vitamins and minerals like vitamin D and iron can help with certain types of hair loss, but more research is needed for others.

The document concluded that FDA-approved treatments like minoxidil and finasteride are effective for hair loss, while the effectiveness of natural remedies and other non-approved treatments is not well-supported by evidence.

Two siblings stayed rickets-free for 14 years after stopping treatment.

Mutations in the Vitamin D receptor gene can cause hair loss similar to mutations in the Hairless gene.

As skin cells mature, vitamin D receptor levels decrease while retinoid X receptor α levels increase.

Some babies are born with alopecia areata, and a treatment with clobetasol propionate can regrow hair in half of the cases.

The document concludes that early diagnosis and treatment are crucial for children with hair loss to prevent permanent damage, although not all conditions can be effectively treated.

Vitamin D might help regulate insulin in the body, but taking Vitamin D supplements doesn't clearly prevent or improve type 2 diabetes. More research is needed.