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- 6874 A Rare Case Of Hereditary 1,25 (OH)2D Resistant Rickets
- SUN-332 A Rare Case Of Hereditary 1,25 (OH)2D Resistant Rickets
- 1,25-(OH)2D3 promotes hair growth by inhibiting NLRP3/IL-1β and HIF-1α/IL-1β signaling pathways
- In vivo microautoradiography of [3H]1,24(OH)2D3 (tacalcitol) following topical application to normal rats and in vitro metabolism in human keratinocytes
- An unusual presentation of vitamin D dependent rickets type 2 with low 25 (OH) D3 levels and alopecia: a case report of two siblings
- 25 Hydroxyvitamin D 1 α-Hydroxylase Is Required for Optimal Epidermal Differentiation and Permeability Barrier Homeostasis
- Hereditary 1,25‐Dihydroxyvitamin D–Resistant Rickets Due to an Opal Mutation Causing Premature Termination of the Vitamin D Receptor
- The vitamin D receptor functions as a transcription regulator in the absence of 1,25-dihydroxyvitamin D3
- Two New Unrelated Cases of Hereditary 1,25-Dihydroxyvitamin D-resistant Rickets with Alopecia resulting from the same Novel Nonsense Mutation in the Vitamin D Receptor Gene
- Evidence for 1,25-Dihydroxyvitamin D3-independent Transactivation by the Vitamin D Receptor
- 211 Effect of 1α,25-dihydroxyvitamin D3 on mechanical properties of hairless mouse skin
- Hereditary 1,25-Dihydroxyvitamin D Resistant Rickets due to a Mutation Causing Multiple Defects in Vitamin D Receptor Function
- Proliferation and Differentiation of Cultured Human Keratinocytes is Modulated by 1,25(OH)<sub>2</sub>D<sub>3</sub> and Synthetic Vitamin D<sub>3</sub> Analogues in a Cell Density‐, Calcium‐ and Serum‐Dependent Manner*
- Vitamin D and Human Health: Lessons from Vitamin D Receptor Null Mice
- Vitamin D metabolism and function in the skin
- Rickets with alopecia: An inborn error of vitaminD metabolism
- Vitamin D and the skin: Physiology and pathophysiology
- Inactivation of the Vitamin D Receptor Enhances Susceptibility of Murine Skin to UV-Induced Tumorigenesis
- Vitamin D-dependent rickets type II. Defective induction of 25-hydroxyvitamin D3-24-hydroxylase by 1,25-dihydroxyvitamin D3 in cultured skin fibroblasts.
- Novel mechanisms for the vitamin D receptor (VDR) in the skin and in skin cancer
- The Vitamin D Receptor as Tumor Suppressor in Skin
- Hereditary vitamin D-resistant rickets (HVDRR) owing to a heterozygous mutation in the vitamin D receptor
- Vitamin D: Role in Skin and Hair
- Vitamin D and the Parenteral Nutrition Patient
- The Molecular Biology of the Vitamin D Receptor
- Vitamin D receptor ablation alters skin architecture and homeostasis of dendritic epidermal T cells
- A human vitamin D receptor mutation causes rickets and impaired Th1/Th17 responses
- Ligand‐Independent Actions of the Vitamin D Receptor: More Questions Than Answers
- Vitamin D3 and chemotherapy-induced alopecia
- Rickets with Alopecia Signals Vitamin D Dependent Rickets Type II