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This study found that selective downregulation of CD200 in the bulge area might contribute to unusual bulge involvement in a subset of alopecia areata cases.

This study suggests that neuropeptides may contribute to the progression of alopecia areata by causing neurogenic inflammation and disrupting the inflammatory environment of hair follicles, but the exact pathogenesis still requires further investigation.

This study explored the immune environment of the hair follicle's bulge region and suggested that its unique signaling may prevent melanoma formation by lacking necessary proinflammatory signals for full oncogenic transformation, indicating potential strategies for melanoma prevention by replicating this immune-privileged environment.

This study reports that in primary cicatricial alopecias, increased expression of certain immune markers in the hair-follicle bulge suggests a collapse of immune privilege, which may contribute to follicle destruction.

This article discusses the anatomical and biological aspects of hair follicle stem cells in the bulge region but reports no new research findings.