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- Insights into the context-dependent immunological roles of the CXCL12–CXCR4 axis in alopecia
- Regulatory T cells in skin utilize the Cxcr4-Cxcl12 axis to promote hair follicle regeneration
- The homing of exogenous hair follicle mesenchymal stem cells into hair follicle niches
- CXCL12 inhibits hair growth through CXCR4
- CXCR3 Blockade Inhibits T Cell Migration into the Skin and Prevents Development of Alopecia Areata
- Author response: Neutrophils promote CXCR3-dependent itch in the development of atopic dermatitis
- Decision letter: Neutrophils promote CXCR3-dependent itch in the development of atopic dermatitis
- CXCL12+ dermal fibroblasts promote neutrophil recruitment and host defense by recognition of IL-17
- Recent Advances in Drug Development for Hair Loss
- Antroquinonol Exerts Immunosuppressive Effect on CD8<sup>+</sup> T Cell Proliferation and Activation to Resist Depigmentation Induced by H<sub>2</sub>O<sub>2</sub>
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