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- Insights into the context-dependent immunological roles of the CXCL12–CXCR4 axis in alopecia
- Regulatory T cells in skin utilize the Cxcr4-Cxcl12 axis to promote hair follicle regeneration
- CXCL12 inhibits hair growth through CXCR4
- CXCR3 Blockade Inhibits T Cell Migration into the Skin and Prevents Development of Alopecia Areata
- Author response: Neutrophils promote CXCR3-dependent itch in the development of atopic dermatitis
- The chemokine SDF-1/CXCL12 regulates the migration of melanocyte progenitors in mouse hair follicles
- CXCL12+ dermal fibroblasts promote neutrophil recruitment and host defense by recognition of IL-17
- Increased Circulating CXCL10 in Non-Segmental Vitiligo Concomitant with Autoimmune Thyroid Disease and Alopecia Areata
- Decision letter: Neutrophils promote CXCR3-dependent itch in the development of atopic dermatitis
- Th1 Chemokine CXCL10 and Alopecia Areata: The Possible Target for the Treatment of Alopecia Areata
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