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- Local circadian clock gates cell cycle progression of transient amplifying cells during regenerative hair cycling
- Clock genes, hair growth and aging
- Mutant laboratory mice with abnormalities in hair follicle morphogenesis, cycling, and/or structure: An update
- Transgenic mice display hair loss and regrowth overexpressing mutant <i>Hr</i> gene
- Timing of expression of the core clock gene <i>Bmal1</i> influences its effects on aging and survival
- Chronic circadian misalignment is a risk factor for hair growth impairment
- Premature aging and cancer development in transgenic mice lacking functional CYLD
- The clock gene brain and muscle Arnt-like protein-1 (BMAL1) is involved in hair growth
- Brain and muscle Arnt-like protein-1 (BMAL1) controls circadian cell proliferation and susceptibility to UVB-induced DNA damage in the epidermis
- Hairless controls hair fate decision via Wnt/β–catenin signaling
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