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This study suggests that human outer root sheath cells are suitable for skin replacement, as they can form a stratified epithelium similar to normal epidermis in organotypic cultures.

This study found that KY19382, a small molecule activating the Wnt/β-catenin pathway, significantly improved wound healing by enhancing cell migration, increasing collagen and stem cell markers, and accelerating re-epithelialization and neo-epidermis formation in a murine model without causing significant cytotoxicity.

Blocking cell death in certain stem cells can improve wound healing and tissue regeneration.

Reducing neutrophils or inhibiting NETs improves wound healing in sickle cell disease.

New therapies for burn wounds show promise in reducing pain, infection risk, and improving healing and physical outcomes.