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AIRE protein, defective in APECED patients, is found in skin and hair cells and interacts with cytokeratin 17.

Notch1 signaling is impaired in hidradenitis suppurativa, affecting skin and hair cells.

Killing specific cells in hair follicles can lead to hair growth problems in mice.

The conclusion suggests that early signs of hidradenitis suppurativa may start in the hair follicle, with genetic and immune factors playing a role, highlighting the need for more research on the hair follicle's immune system.

Hair graying is caused by the loss of pigment cells due to poor maintenance of stem cells in the hair follicle.

Genetic mutations can cause hair growth disorders by affecting key genes and signaling pathways.

Adiponectin reduces inflammation and bone loss in joint replacements.

Hair growth issues can be linked to genetics, diseases, or medications, and new treatments are being developed.

Msx2 deficiency in mice leads to bone growth and organ development problems.

The new method using gene-modified stem cells and a 3D printed scaffold improved skin repair in mice.

The document says that treating the root cause of hair follicle damage is crucial to prevent permanent hair loss, and treatment options vary.

A new method using special materials can help regrow hair by creating small wounds.

Hair loss in certain mice is linked to changes in keratin-related genes.

4-META resin heals skin wounds faster and better than cyanoacrylate.

Hair greying is caused by oxidative stress damaging hair follicles and melanocytes.

The ITGB6 gene is important for tissue repair and hair growth, and mutations can lead to enamel defects and other health issues.

EGFR helps control how hair grows and forms without needing p53 protein.

Radish saponin extract may help grow hair in balding mice.

MIM is crucial for hair follicle formation and regeneration by controlling cilia formation and hedgehog signaling through its interaction with Cortactin and Src.

A wearable device using electric stimulation can significantly improve hair growth.

Hidradenitis suppurativa is a skin condition possibly caused by hair follicle issues, with treatments focusing on infection and inflammation.

mTOR signaling helps activate hair stem cells by balancing out the suppression caused by BMP during hair growth.

The conclusion is that Fgf18 and Tgf-ß signaling could be targeted for hair loss treatments.

Hair loss can indicate underlying systemic diseases and addressing these can sometimes reverse the hair loss.

Damaged hair follicle stem cells can cause permanent hair loss, but understanding their role could lead to new treatments.

Hair follicles are essential for skin health, aiding in hair growth, wound healing, and immune function.

Overexpressing ovine β-catenin in mice skin increases hair follicle density and growth.

Skin organoids with NCSTN mutation show changes in hair follicle development and higher inflammation, key features of Hidradenitis Suppurativa.

Uncombable hair syndrome causes frizzy hair and can affect the nervous system, eyes, and ears, often co-occurring with other hair, skin, nail, and teeth conditions, and is linked to three specific gene mutations.

A variant in the ADAM17 gene causes hair loss by increasing protein degradation through TRIM47.