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Finasteride effectively reduces hair growth in women with idiopathic hirsutism, but requires careful contraception during treatment.

Finasteride slowly binds to 5-alpha-reductase, affecting enzyme stability and inhibitor potency.

5α-reductase inhibitors and alpha-1 adrenergic antagonists together effectively treat benign prostatic hyperplasia, with long-term benefits.

Finasteride potentially treats hair loss by reducing DHT production.

The medications 5α-reductase inhibitors and spironolactone are generally safe for breast cancer patients on endocrine therapies and do not significantly increase breast cancer risk.

GI198745 is more potent and longer-lasting than finasteride, potentially better for treating hair loss.

FCE 28260 is a stronger and longer-lasting inhibitor of 5α-reductase than finasteride, which may make it a better treatment for certain medical conditions.

Finasteride boosts morphine's pain relief, stops tolerance, and reduces withdrawal in rats.

Finasteride doesn't affect erections much, but may decrease libido in men.

The enzyme type 1 5α-reductase is more active in the hair follicle's lower part than in the skin's outer layer.

The main enzyme found in pubic skin that could be targeted to treat excessive hair growth is 5 alpha-R2.

5α-reductase inhibitors like finasteride may not be effective or safe for transgender individuals, and more research is needed.

New compounds were made that effectively block a specific enzyme related to androgen conditions.

5α-reductase inhibitors can interfere with doping tests by masking banned substances.

Reducing 5α-reductase activity helps endometrial cells differentiate, aiding pregnancy.

The PW91 method is best for calculating the basic physical and chemical properties of Finasteride, a drug used for prostate issues and hair loss.

HST and HST/F promote hair growth and may help treat alopecia.

Finasteride's sexual side effects not caused by androgen deficiency or SRD5A inhibition.

5α-reductase inhibitors may prevent prostate cancer but could also raise the risk of more severe cancers.

Finasteride safely lowers DHT levels without affecting testosterone.

Finasteride treats enlarged prostate and hair loss, but may cause side effects in some patients.

Finasteride effectively blocks enzyme causing male pattern baldness.

Male pattern baldness involves hormones and cell signals affecting hair growth.

Finasteride affects male rat genitalia development, causing abnormalities during specific pregnancy days.

Four-amino acid part makes enzyme sensitive to finasteride.

A three-month treatment with a GnRH agonist significantly lowered androgen levels and 5α-reductase activity in men with benign prostatic hyperplasia.

Finasteride reduces hairiness and androgen levels in women with unexplained excessive hair growth.

Hair sensitivity to androgens is partly controlled by specific enzyme expressions in different hair areas.

Finasteride reduces scalp DHT levels, potentially treating male pattern baldness.

A49T gene variant linked to higher prostate cancer risk, lower hormone levels, and slightly reduced balding risk.