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AHCC reduces hair loss and liver injury caused by chemotherapy in rodents.

sPLA2-IIA increases growth in hair follicle stem cells and cancer cells, suggesting it could be targeted for hair growth and cancer treatment.

ILC1-like cells can cause alopecia areata by attacking hair follicles.

Prostate cancer cells can make their own androgens to activate the androgen receptor, and treatments like abiraterone may increase this ability, suggesting new therapies should target the entire steroid-making pathway.

SKPs are similar to adult skin stem cells and could help in skin repair and hair growth.

Blocking JAK-STAT signaling can lead to hair growth.

Cancer can hijack the body's cell repair system to promote tumor growth, and targeting this process may improve cancer treatments.

Lymphocytes, especially CD8+ T cells, play a key role in causing alopecia areata, and targeting them may lead to new treatments.

Brain hormones significantly affect hair color and could potentially be used to prevent or reverse grey hair.

Nanoparticles show promise for better wound healing, but more research is needed to ensure safety and effectiveness.

The gene Msx2 is crucial for hair follicle regeneration during wound healing.

A protein called EGFR protects hair follicle stem cells, and when it's disrupted, hair follicles can be damaged, but blocking certain pathways can restore hair growth.

Regulatory T-cells are important for healing and regenerating tissues in various organs by controlling immune responses and aiding stem cells.

Conditioned media from mesenchymal stem cell cultures could be a more effective alternative for regenerative therapies, but more research is needed.

The document concludes that alopecia has significant social and psychological effects, leading to a market for hair loss treatments.

Different body areas in mice produce different hair types due to interactions between skin layers.

Overactivating Hedgehog signaling makes hair follicle cells in mice grow hair faster and create more follicles.

YAP1 is important for skin regeneration and may affect skin disorder treatments.

CD8+ T cells are involved in alopecia areata and may cause disease relapse.

Hair follicle stem cells are controlled by their surrounding environment.

ILC1-like cells can independently cause alopecia areata by affecting hair follicles.

Targeting specific T cells may help treat alopecia areata.

Alopecia areata can be reversed by JAK inhibitors, promoting hair regrowth.

Low-dose radiation affects hair stem cell function and survival by changing their genetic material's structure.

Stopping mitochondrial respiration can prevent brain cancer spread in skin cancer patients, and plant compound β-sitosterol could help achieve this.

iPSC-derived stem cells on a special membrane can help repair full-thickness skin defects.

Hair follicles have a more inactive cell cycle than other skin cells, which may help develop targeted therapies for skin diseases and cancer.

RANKL causes lymph nodes to grow by making certain cells multiply.

The review suggests that a special cell-derived treatment shows promise for various skin conditions and hair growth but needs more research for confirmation.

The document concludes that understanding and treatments for alopecia areata have significantly advanced, now recognizing it as an autoimmune disorder.