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Mice genetically modified to produce more CD109 in their skin had less inflammation and better healing with less scarring.

Blood cells turned into stem cells can become skin cells similar to normal ones, potentially helping in skin therapies.

Some wound-healing cells can turn into fat cells around new hair growth in mice.

Estrogen is important for keeping adult mouse nipple skin healthy by controlling certain cell signals.

The p53 protein has complex, sometimes contradictory functions, including tumor suppression and promoting cell survival.

Future hair loss treatments should aim to extend hair growth, reactivate resting follicles, reverse shrinkage, and possibly create new follicles, with gene therapy showing promise.

The research found that different types of fibroblasts are involved in wound healing and that some blood cells can turn into fat cells during this process.

Spiny mice are better at regenerating hair after injury than laboratory mice and could help us understand how to improve human skin repair.

Blocking the Wnt/β‐catenin pathway can speed up wound healing, reduce scarring, and improve cartilage repair.

ZO-1 helps hair follicle stem cells renew better by changing their structure.

Glutamine metabolism affects hair stem cell maintenance and their ability to change back to stem cells.

Layer-by-Layer self-assembly is promising for biomedical uses like tissue engineering and cell therapy, but challenges remain in material safety and process optimization.

Platelet-rich plasma may improve wound healing by stimulating cell growth and blood vessel formation.

Mice with enhanced regeneration abilities may help develop new regenerative medicine therapies.

Changing the environment around stem cells could help tissue repair, but it's hard to be precise and avoid side effects.

BLIMP1 is essential for skin maintenance but not for defining sebaceous gland progenitors.

Alkaline Ceramidase 1 prevents early hair loss in mice by keeping hair follicle stem cells balanced.

3D cell-derived matrices improve tissue regeneration and disease modeling.

New methods to improve the healing abilities of mesenchymal stem cells for disease treatment are promising but need more research.

B2m-free HLA variants may be a new class of HLA important in immune responses and diseases.

Disulfide bonds in keratin fibers break more easily under stress, especially when wet, affecting fiber strength.

Cold atmospheric plasma may speed up wound healing and control infections.

Touch sensitivity in mouse skin decreases during hair growth due to changes in touch receptors.

Epigenetic mechanisms control skin development by regulating gene expression.

The PIP5K1A gene helps cashmere growth in goats by promoting cell proliferation, and melatonin boosts its expression.

The shape of fibrous scaffolds can improve how stem cells help heal skin.

Human hair keratins can self-assemble and support cell growth, useful for biomedical applications.

The gene p53 is crucial for removing damaged cells to allow for healthy tissue renewal.

The immune system plays a key role in tissue repair, affecting both healing quality and regenerative ability.

High levels of ERK activity are key for tissue regeneration in spiny mice, and activating ERK can potentially redirect scar-forming healing towards regenerative healing in mammals.